What is the logic behind taking nolva AND aromasin? Think about it, post cycle natural test is almost non existant and aromasin will make sure there is little or not conversion to E. So then you take nolva which binds to the E receptor and makes sure what little E you have, if any, has no where to bind. seems like overkill to me.
Then look at what HCG does, its triggers the testes into producing test. if your on say a 12 week cycle your testes havent done anything for 12 weeks so when you stop taking test your body is going to try and trigger test production.
Lets put this another way. Say you couldnt lift heavy for 12 weeks but you could lift light 2x a week, not enough weight to cause any muscle gains but enough to help preserve what you have. Would you chose to not exercise at all or workout lightly a couple times a week?
If you would opt for the couple light workouts a week, why? Guess what, taking HCG a couple times a week at light doses is like a light workout for your testes while on cycle.
Agreed, the entire point of PCT is to get the hypothalamus to start releasing GnRH once again.
GnRH is the signal responsible for the pituitary release of LH, which is responsible for raising endogenous testosterone which can then be converted to estrogen.
When we cycle, the presence of excess estrogens and androgens is recognized by the brain and, in an effort to reach homeostasis, the hypothalamus stops GnRH signaling (to try and lower hormones).
Nolvadex, Clomid, and Aromasin can all theoretically achieve the same desired resposne by significantly lowering estrogen, or blocking receptors, fooling the brain into thinking that hormone levels are almost non-existent - triggering the release of GnRH->LH->Testosterone.
A lot of people have the misconception that they need to shotgun PCT or hit it as hard as they would a cycle, and this is not the case at all. This is why so many people suffer from horrible emotional and libido related side effects when taking SERMs like nolvadex or clomid or even combining a handful of different things for PCT. Completely unnecessary.
One drug that either eliminates estrogen or blocks estrogen receptors is enough.
A low dose of these any of these pharmacuiticals should be plenty enough to block the low estrogen levels one would have when they stop cycling (low endogenous testosterone = low estrogen).
Users of steroids understand the need for PCT - because their HPTA is suppressed. This understanding needs to be carried over during the consideration of PCT drug quantities. Why use a high dose SERM if you know your natural testosterone production is already very low (meaning low estrogen).
Anyone that is planning on cycling for a long period of time, and set on using drugs like clomid, nolvadex, etc for PCT should put some effort into finding their own sweet spot with respect to the dosage of the drug (where LH and testosterone are at the top of the range). This could significantly reduce or prevent the side effects that some have experienced.
The use of low dose HCG is also important to consider while cycling (especially when cycling for long periods). When the testes are not receiving LH they don't just stop the production of testosterone. LH is also responsible for increasing the activity of the P450scc enzyme, which is responsible for the conversion of cholesterol to pregnenolone. Pregnenolone is the mother hormone / primary building block for all other steroid hormones in the body. HCG mimics LH in the testes, keeping this enzyme active.