Flexx said:
I hit the clomid two weeks after I finished and I'm back to normal. No more tenderness or that extreme puffiness.
2 weeks afterward, your tenderness and puffiness may have subsided anyway.
Side effects of clomid:
blurred vission (but mostly in women), diplopia, scotoma, photo-phobia, nausea, vomiting, bloating, distention, water gain, breast discomfort, hot flashes, urinary infections, cyst formation, thrombocytopenia, leukopenia, anemia, pharyngitis, rhinitis, sinusitus, epistaxis, dyspnea, headaches, restlessness. insomnia, dizziness, light-headedness, depression, fatigue...
Clomid is, however, a VERY effective drug in promoting HPTA recovery post-cycle. Due to a feedback inhibition, clomid will not stimulate any natural testosterone production as long as androgen levels in the body remain high. But, post cycle, when androgen levels fall toward normal, clomid will bind to receptors in the hypothalamus and trick the pituatary into releaseing more LH... LH stimulates the testicles to produce testosterone, and testosterone is converted to estrogen. Anytime the body gets low in estrogen, it must make more testosterone in order to increase estrogen levels. Estrogen is absolutely necessary in various cellular and neural functions such as the branching of dendritic spines (memory)....
Use Arimidex or Proviron as antiestrogens throughout your cyle as antiestrogens. It is doubtful that clomid will have any benefit in preventing gyno... and will not prevent aromitase from converting testosterone into estrogen where proviron and arimidex will.
Arimidex is VERY effective and doesn't cost much more than other antiestrogens if you buy overseas (it is not controlled so it gets through customs).
Nolvadex can be used throughout a cycle and is effective at blocking estrogen receptors but is useless at preventing estrogen's formation. Nolvadex will also inhibit IGF-1 and GH which could severly effect your cycle... High levels of androgens normally cause an increase in IGF-1 mediated growth responses by increasing IGF-1 receptor concentration and hormone levels. This is the same pathway that GH uses... block the IGF-1 pathway with nolvadex, and you will not make the gains that you could have.
Given, there are those who say they gained just fine while using nolvadex, but truth be told, they would have gained a lot more had they researched.
Proviron is also good to use throughout. Proviron, however, will also elicit an AR-mediated response meaning it will cause some degree of anabolism in itself. Consequently, the anabolic response from proviron is not as strong as the anabolic response from the androgens that you are running it with (i.e. test, nandralone, etc). Thus, by competing with the same receptors, it could possibly decrease the overall effects of the primary anabolics.
BUT this effect will be VERY small if at all... the reason is that androgen receptors tend to have different affinities (binding abilities) to different gear. For instance, androgen receptors have a higher affinity towards DHT than they do testosterone. It is unlikely even though proviron has an AR-mediated response, that it will bind to the androgen receptors as redially as the other androgens that your cycle consists of. But aromitase enzymes DO have an affinity toward proviron, so this lowers the oveall competitive inhibition with androgens for androgen receptors.
Arimidex is the drug of choice... Dosses of 1/4 tab per day have shown to be extremely effective. There was almost no difference in the effects of 1mg/day and 7mg/day.... There is no point in going over 1/2 tab per day IMO unless you are running over 1.5g of testosterone per week. 28 tabs come in a box... The price of Arimidex has dropped signifigantly due to new products that contain anastrozole, the active ingredient in Arimidex.
-Stew
-Stew
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