Please Scroll Down to See Forums Below psychedout
New member
I have been trying to find it and am coming up empty handed...
Can you or someone else post it...
Thanks.
Can you or someone else post it...
Thanks.
Fonz... whats the link for you dbol bridge theory...
- Thread starter psychedout
- Start date
DeepZenPill
New member
Since I just flamed Fonz on another thread... here you go:
http://www.massmonsters.com/6/ubb.x?a=tpc&s=9506025911&f=9966039821&m=564600241
I think there's registration required (and I'm the one who always bitches about that), but I think the same article here on EF is now in the Platinum section.
http://www.massmonsters.com/6/ubb.x?a=tpc&s=9506025911&f=9966039821&m=564600241
I think there's registration required (and I'm the one who always bitches about that), but I think the same article here on EF is now in the Platinum section.
Rio 2001
New member
Absolute Final Version:
(Also posted at EF and CEM)
The Dball Main cycle ramp-off explained in detail: Article (post #1)
The original Study:
Acta Endocrinol (Copenh) 1976 Dec;83(4):856-64 Related Articles, Links
Effect of an anabolic steroid (metandienon) on plasma LH-FSH, and testosterone and on the response to intravenous administration of LRH.
Holma P, Adlercreutz H.
Plasma levels of testosterone, luteinizing hormone (LH) and follicle-stimulating hormone (FSH) as well as the response of LH and FSH to the intravenous administration of 100 mug of luteinizing hormone releasing hormone (LRH) were measured in 16 well-trained athletes (mean age 30 years) before and after 2 months of daily oral intake of 15 mg of metandienon, and anabolic steroid (Anabolin, 17 alpha-methyl-17beta-hydroxy-1,4-androstadien-3-one, Medica, Finland). All athletes continued to train regularly, just as they had done for several years. During administration of metandienon the mean plasma testosterone level fell 69%, from 29.4 +/- 11.6 nmol/1 to 9.1 +/- 7.5 nmol/1. The mean plasma levels of LH and FSH also fell significantly (P less than 0.001 and P less than 0.01, respectively), both about 50%. Because LH and FSH levels were low after administration of the steroid the maximum stimulation values after LRH administration were also lower than pre-treatment values although the mean increments did not differ significantly before and after administration of the anabolic steroid. However, after treatment, the FSH response curve had a biphasic pattern in most subjects, with peaks at 10 to 20 and 50 to 60 min after the iv injection of LRH. Administration of LRH after the treatment period had no effect on FSH secretion in two subjects and no effect on LH secretion in one. Our results show that administration of an anabolic steroid causes a pronounced lowering of plasma levels of testosterone, LH and FSH but causes no gross alteration in the response of LH secretion to stimulation by LRH. The reason for the biphasic response pattern of FSH to LRH administration in most subjects is not known.
------------------------------------------------------------------------------------
Ok, I'm making this flame-free so that no flame fests occurr and people get the right information, not all jumbled. It has actually been improved upon somewhat with the advent of some newer compounds and with the help of AF Vets.
This a more streamlined version, that should work for almost everybody.(Even those who almost always crash):
First, it was decided that the last 4 weeks, of the 8 week ramp-off should be 5mg instead of 10mg, as your HPTA should be recovered by some degree by that time.
The 10mg time/correlation:
Now, for 10mg.
10mg ------- 5mg --------- 2.5mg --------1.25mg ---------0.625mg
Half-lives = 3.4 * 4 = 13.6hrs(to 1mg)
13.6/16.0 = 85% or 0.85(As compared to 15mg)
Test decrease + LH Decrease
Normal(15mg) = 69% 50%
10mg = 58.7% 42.5%
Thats w/o arimidex
W/ arimidex(Increases Test by an average of 58% in controlled trails, and reduces estrogen by about 90%):
10mg becomes: 58.7*(1-0.58)= 24.65% Test decrease
LH is reduction is reduced from 50% to 42.5%
(These numbers do not include the addition of Clomid and ***)
This is 5mg time/concentration correlation:
5mg ------- 2.5mg --------1.25mg ------ 0.625mg
Half-lives = 2.4 * 4 = 9.6hrs(to 1mg)
(Compared to 10mg)
9.6/13.6 = 70.59% or 0.7059
Test decrease + LH Decrease
Normal(15mg) = 69% 50%
10mg = 58.7%(-10.3%) 42.5%(-7.5%)
5mg = 41.43%(-27.57%) 30%(-12.5%)
Thats w/o arimidex
W/ arimidex:
5mg becomes: 41.43*(1-0.58)= 17.4% Test decrease
And LH, decreases to only a 30% decrease
(These numbers do not include the addition of Clomid or ***)
Finalizing(W/arimidex included):
Testosterone decrease + LH Decrease
15mg == 69% 50%(Study)
10mg == 24.65% 42.5%(This for you weeks 1-4)
5mg == 17.4% 30%(This for you weeks 5-8)
(These numbers are in comparison to the 15mg dosage and do not include the addition of Clomid or *** or other compounds which both stimulate LH, FSH and endo test)
End(Finalized Ramp-off):
(6-8 weeks) before end of Main cycle: Start 25mg Proviron
End Main Cycle.
AM Dball off-ramp: 8 weeks
#1.Start ramp-off at 10mg Dball in the AM upon
waking up.(Weeks 1-4) and 5mgs(Weeks 5-8)
#2 Make damn sure you take the 10mg dball(Weeks 1-4) at the same damn time every day. As soon as you wake up. This wake up time (if 8 or 9 or 10 AM) must be used for the rest of the ramp-off(8 weeks)(Circadian Rhythm is VERY important to
the success of the ramp-off) Same with the 5mg dose(Weeks 5-8).
#3 Proviron at 25mgs/day(LH booster)(Weeks 1-8)
#4 Arimidex at 1mg ED or more.(2mg is as high
as I would go).(Also on weeks 1-8)
#5 *** at 5000IU’s 2X/week on Weeks 5,6,7,8
(Endo Test Booster)
#6 Clomid at 300mgs Day 1, and then 100mgs/day from then on until the end of the bridge(LH and FSH Booster)
Weeks 1-4: Are designed to reduce androgen levels(10mg Dball here) and stimulate the HPTA to start its own endo test production, and increase FSH, and LH levels.
10mg == 24.65% 42.5%(This for you weeks 1-4)
Your HPTA will be pretty much zero at the beginning off the ramp-off, and a combination of compounds that only decrease normal test levels by 24.65% and substances that increase LH and FSH and stimulate endo test(Clomid and Proviron) will let your HPTA recover by some margin.
Weeks 5-8: Are designed to reduce androgen levels even further(Only 5mg dball here), due to the fact that they are not needed because your HPTA has partially recovered in Weeks 1-4.
5mg == 17.4% 30%(This for you weeks 5-8)
With the reduction of androgens from 10mg to 5mg, you have changed the Test decrease to only 17.4% and let LH reduction only be 30%. Again, you will be on Clomid and Proviron, and these two will help you boost LH levels and increase test levels further. But now comes the interesting part. The introduction of ***. *** is probably the best booster of endo test at your disposal for short periods of time, via the stimulation of the Leydig Cells in the testes. *** at the specified dosage will boost endo test levels back to the normal pre-main cycle range. (Remember that 5mg dball only causes a 17.4% decrease in Test).
*** should help nullify that.
And after those 8 weeks of the ramp-off, you come off. Both Test and LH levels should be within the normal range, and you will not crash or suffer muscle loss like pretty much all post-cycle recovery methods.
Special thanks goes to the guys who helped me put all this into a clear and concise article. You know who you are.
NEW:
As an addition: Androgel might be of definate use in Weeks 5-8 as it roughly doubles Test levels, w/o any decrease in LH. Which will further boost ending ramp-off test levels. If you have the time, this preparation shows great promise for further enhancing the ramp-off.
References:
(Thanks to Mr. Nobody and got Wood)
1. Swerdloff, RS, and Wang, C., et al., Long-term pharmacokinetics of transdermal testosterone gel in hypogonadal men., J Clin Endocrinol Metab. 2000 Dec;85(12):4500-10.
The intend of agel in the recovery program is for libido purposes, and may delay overall recovery a bit. However, its not just about fast recovery but achieving homeostasis in a managable manner, without slipping into depression and muscle loss while you wait for endo test to come back. The same goes for morning dbol.
As everyone knows already, only time will recover you completely.
from Gotwood:
testo gel lowered LH but not too much
Got Wood? note : these men ranged from 22-65. Testo enanthate (inj) lowered LH too much - to subnormal values. Testo gel lowered LH to normal, but not beyond to a subnormal range. This is evidence that the testo gel may not lower the LH too much, thereby inhibiting recovery. however again these are hypogonadal men.
============================
J Clin Endocrinol Metab 1999 Oct;84(10):3469-78
Pharmacokinetics, efficacy, and safety of a permeation-enhanced testosterone transdermal system in comparison with bi-weekly injections of testosterone enanthate for the treatment of hypogonadal men.
Dobs AS, Meikle AW, Arver S, Sanders SW, Caramelli KE, Mazer NA.
Johns Hopkins Medical Center, Baltimore, Maryland 21287, USA. [email protected]
The pharmacokinetics, efficacy, and safety of the Androderm testosterone (T) transdermal system (TTD) and intramuscular T enanthate injections (i.m.) for the treatment of male hypogonadism were compared in a 24-week multicenter, randomized, parallel-group study. Sixty-six adult hypogonadal men (22-65 years of age) were withdrawn from prior i.m. treatment for 4-6 weeks and then randomly assigned to treatment with TTD (two 2.5-mg systems applied nightly) or i.m. (200 mg injected every 2 weeks); there were 33 patients per group. Twenty-six patients in the TTD group and 32 in the i.m. group completed the study. TTD treatment produced circadian variations in the levels of total T, bioavailable T, dihydrotestosterone, and estradiol within the normal physiological ranges. i.m. treatment produced supraphysiological levels of T, bioavailable T, and estradiol (but not dihydrotestosterone) for several days after each injection. Mean morning sex hormone levels were within the normal range in greater proportions of TTD patients (range, 77-100%) than i.m. patients (range, 19-84%). Both treatments normalized LH levels in approximately 50% of patients with primary hypogonadism; however, LH levels were suppressed to the subnormal range in 31% of i.m. patients vs. 0% of TTD patients. Both treatments maintained sexual function (assessed by questionnaire and Rigiscan) and mood (Beck Depression Inventory) at the prior treatment levels. Prostate-specific antigen levels, prostate volumes, and lipid and serum chemistry parameters were comparable in both treatment groups. Transient skin irritation from the patches was reported by 60% of the TTD patients, but caused only three patients (9%) to discontinue treatment. i.m. treatment produced local reactions in 33% of patients and was associated with significantly more abnormal hematocrit elevations (43.8% of patients) compared with TTD treatment (15.4% of patients). Gynecomastia resolved more frequently during TTD treatment (4 of 10 patients) than with i.m. treatment (1 of 9 patients). Although both treatments seem to be efficacious for replacing T in hypogonadal men, the more physiological sex hormone levels and profiles associated with TTD may offer possible advantages over i.m. in minimizing excessive stimulation of erythropoiesis, preventing/ameliorating gynecomastia, and not over-suppressing gonadotropins
(Also posted at EF and CEM)
The Dball Main cycle ramp-off explained in detail: Article (post #1)
The original Study:
Acta Endocrinol (Copenh) 1976 Dec;83(4):856-64 Related Articles, Links
Effect of an anabolic steroid (metandienon) on plasma LH-FSH, and testosterone and on the response to intravenous administration of LRH.
Holma P, Adlercreutz H.
Plasma levels of testosterone, luteinizing hormone (LH) and follicle-stimulating hormone (FSH) as well as the response of LH and FSH to the intravenous administration of 100 mug of luteinizing hormone releasing hormone (LRH) were measured in 16 well-trained athletes (mean age 30 years) before and after 2 months of daily oral intake of 15 mg of metandienon, and anabolic steroid (Anabolin, 17 alpha-methyl-17beta-hydroxy-1,4-androstadien-3-one, Medica, Finland). All athletes continued to train regularly, just as they had done for several years. During administration of metandienon the mean plasma testosterone level fell 69%, from 29.4 +/- 11.6 nmol/1 to 9.1 +/- 7.5 nmol/1. The mean plasma levels of LH and FSH also fell significantly (P less than 0.001 and P less than 0.01, respectively), both about 50%. Because LH and FSH levels were low after administration of the steroid the maximum stimulation values after LRH administration were also lower than pre-treatment values although the mean increments did not differ significantly before and after administration of the anabolic steroid. However, after treatment, the FSH response curve had a biphasic pattern in most subjects, with peaks at 10 to 20 and 50 to 60 min after the iv injection of LRH. Administration of LRH after the treatment period had no effect on FSH secretion in two subjects and no effect on LH secretion in one. Our results show that administration of an anabolic steroid causes a pronounced lowering of plasma levels of testosterone, LH and FSH but causes no gross alteration in the response of LH secretion to stimulation by LRH. The reason for the biphasic response pattern of FSH to LRH administration in most subjects is not known.
------------------------------------------------------------------------------------
Ok, I'm making this flame-free so that no flame fests occurr and people get the right information, not all jumbled. It has actually been improved upon somewhat with the advent of some newer compounds and with the help of AF Vets.
This a more streamlined version, that should work for almost everybody.(Even those who almost always crash):
First, it was decided that the last 4 weeks, of the 8 week ramp-off should be 5mg instead of 10mg, as your HPTA should be recovered by some degree by that time.
The 10mg time/correlation:
Now, for 10mg.
10mg ------- 5mg --------- 2.5mg --------1.25mg ---------0.625mg
Half-lives = 3.4 * 4 = 13.6hrs(to 1mg)
13.6/16.0 = 85% or 0.85(As compared to 15mg)
Test decrease + LH Decrease
Normal(15mg) = 69% 50%
10mg = 58.7% 42.5%
Thats w/o arimidex
W/ arimidex(Increases Test by an average of 58% in controlled trails, and reduces estrogen by about 90%):
10mg becomes: 58.7*(1-0.58)= 24.65% Test decrease
LH is reduction is reduced from 50% to 42.5%
(These numbers do not include the addition of Clomid and ***)
This is 5mg time/concentration correlation:
5mg ------- 2.5mg --------1.25mg ------ 0.625mg
Half-lives = 2.4 * 4 = 9.6hrs(to 1mg)
(Compared to 10mg)
9.6/13.6 = 70.59% or 0.7059
Test decrease + LH Decrease
Normal(15mg) = 69% 50%
10mg = 58.7%(-10.3%) 42.5%(-7.5%)
5mg = 41.43%(-27.57%) 30%(-12.5%)
Thats w/o arimidex
W/ arimidex:
5mg becomes: 41.43*(1-0.58)= 17.4% Test decrease
And LH, decreases to only a 30% decrease
(These numbers do not include the addition of Clomid or ***)
Finalizing(W/arimidex included):
Testosterone decrease + LH Decrease
15mg == 69% 50%(Study)
10mg == 24.65% 42.5%(This for you weeks 1-4)
5mg == 17.4% 30%(This for you weeks 5-8)
(These numbers are in comparison to the 15mg dosage and do not include the addition of Clomid or *** or other compounds which both stimulate LH, FSH and endo test)
End(Finalized Ramp-off):
(6-8 weeks) before end of Main cycle: Start 25mg Proviron
End Main Cycle.
AM Dball off-ramp: 8 weeks
#1.Start ramp-off at 10mg Dball in the AM upon
waking up.(Weeks 1-4) and 5mgs(Weeks 5-8)
#2 Make damn sure you take the 10mg dball(Weeks 1-4) at the same damn time every day. As soon as you wake up. This wake up time (if 8 or 9 or 10 AM) must be used for the rest of the ramp-off(8 weeks)(Circadian Rhythm is VERY important to
the success of the ramp-off) Same with the 5mg dose(Weeks 5-8).
#3 Proviron at 25mgs/day(LH booster)(Weeks 1-8)
#4 Arimidex at 1mg ED or more.(2mg is as high
as I would go).(Also on weeks 1-8)
#5 *** at 5000IU’s 2X/week on Weeks 5,6,7,8
(Endo Test Booster)
#6 Clomid at 300mgs Day 1, and then 100mgs/day from then on until the end of the bridge(LH and FSH Booster)
Weeks 1-4: Are designed to reduce androgen levels(10mg Dball here) and stimulate the HPTA to start its own endo test production, and increase FSH, and LH levels.
10mg == 24.65% 42.5%(This for you weeks 1-4)
Your HPTA will be pretty much zero at the beginning off the ramp-off, and a combination of compounds that only decrease normal test levels by 24.65% and substances that increase LH and FSH and stimulate endo test(Clomid and Proviron) will let your HPTA recover by some margin.
Weeks 5-8: Are designed to reduce androgen levels even further(Only 5mg dball here), due to the fact that they are not needed because your HPTA has partially recovered in Weeks 1-4.
5mg == 17.4% 30%(This for you weeks 5-8)
With the reduction of androgens from 10mg to 5mg, you have changed the Test decrease to only 17.4% and let LH reduction only be 30%. Again, you will be on Clomid and Proviron, and these two will help you boost LH levels and increase test levels further. But now comes the interesting part. The introduction of ***. *** is probably the best booster of endo test at your disposal for short periods of time, via the stimulation of the Leydig Cells in the testes. *** at the specified dosage will boost endo test levels back to the normal pre-main cycle range. (Remember that 5mg dball only causes a 17.4% decrease in Test).
*** should help nullify that.
And after those 8 weeks of the ramp-off, you come off. Both Test and LH levels should be within the normal range, and you will not crash or suffer muscle loss like pretty much all post-cycle recovery methods.
Special thanks goes to the guys who helped me put all this into a clear and concise article. You know who you are.
NEW:
As an addition: Androgel might be of definate use in Weeks 5-8 as it roughly doubles Test levels, w/o any decrease in LH. Which will further boost ending ramp-off test levels. If you have the time, this preparation shows great promise for further enhancing the ramp-off.
References:
(Thanks to Mr. Nobody and got Wood)
1. Swerdloff, RS, and Wang, C., et al., Long-term pharmacokinetics of transdermal testosterone gel in hypogonadal men., J Clin Endocrinol Metab. 2000 Dec;85(12):4500-10.
The intend of agel in the recovery program is for libido purposes, and may delay overall recovery a bit. However, its not just about fast recovery but achieving homeostasis in a managable manner, without slipping into depression and muscle loss while you wait for endo test to come back. The same goes for morning dbol.
As everyone knows already, only time will recover you completely.
from Gotwood:
testo gel lowered LH but not too much
Got Wood? note : these men ranged from 22-65. Testo enanthate (inj) lowered LH too much - to subnormal values. Testo gel lowered LH to normal, but not beyond to a subnormal range. This is evidence that the testo gel may not lower the LH too much, thereby inhibiting recovery. however again these are hypogonadal men.
============================
J Clin Endocrinol Metab 1999 Oct;84(10):3469-78
Pharmacokinetics, efficacy, and safety of a permeation-enhanced testosterone transdermal system in comparison with bi-weekly injections of testosterone enanthate for the treatment of hypogonadal men.
Dobs AS, Meikle AW, Arver S, Sanders SW, Caramelli KE, Mazer NA.
Johns Hopkins Medical Center, Baltimore, Maryland 21287, USA. [email protected]
The pharmacokinetics, efficacy, and safety of the Androderm testosterone (T) transdermal system (TTD) and intramuscular T enanthate injections (i.m.) for the treatment of male hypogonadism were compared in a 24-week multicenter, randomized, parallel-group study. Sixty-six adult hypogonadal men (22-65 years of age) were withdrawn from prior i.m. treatment for 4-6 weeks and then randomly assigned to treatment with TTD (two 2.5-mg systems applied nightly) or i.m. (200 mg injected every 2 weeks); there were 33 patients per group. Twenty-six patients in the TTD group and 32 in the i.m. group completed the study. TTD treatment produced circadian variations in the levels of total T, bioavailable T, dihydrotestosterone, and estradiol within the normal physiological ranges. i.m. treatment produced supraphysiological levels of T, bioavailable T, and estradiol (but not dihydrotestosterone) for several days after each injection. Mean morning sex hormone levels were within the normal range in greater proportions of TTD patients (range, 77-100%) than i.m. patients (range, 19-84%). Both treatments normalized LH levels in approximately 50% of patients with primary hypogonadism; however, LH levels were suppressed to the subnormal range in 31% of i.m. patients vs. 0% of TTD patients. Both treatments maintained sexual function (assessed by questionnaire and Rigiscan) and mood (Beck Depression Inventory) at the prior treatment levels. Prostate-specific antigen levels, prostate volumes, and lipid and serum chemistry parameters were comparable in both treatment groups. Transient skin irritation from the patches was reported by 60% of the TTD patients, but caused only three patients (9%) to discontinue treatment. i.m. treatment produced local reactions in 33% of patients and was associated with significantly more abnormal hematocrit elevations (43.8% of patients) compared with TTD treatment (15.4% of patients). Gynecomastia resolved more frequently during TTD treatment (4 of 10 patients) than with i.m. treatment (1 of 9 patients). Although both treatments seem to be efficacious for replacing T in hypogonadal men, the more physiological sex hormone levels and profiles associated with TTD may offer possible advantages over i.m. in minimizing excessive stimulation of erythropoiesis, preventing/ameliorating gynecomastia, and not over-suppressing gonadotropins
Rio 2001
New member
The Dianabol Bridge Explained (post #1)
I've been reading some of the posts regarding this
bridge and some of them are truly from left-field.
First of, this is a BRIDGE. OK? a B-R-I-D-G-E.
Your LH function and Test levels are supposed
to RECOVER.
Ok, now having said that.
Here's the pharmo-kinetics behind Methandrostenelone,
brand name Dianabol.
10mg taken at once will increase your average testosterone level by 5 times and decrease your endogeneous cosrtisone
by 50-70%.
The reason why dianabol is a good choice for a bridge is that
its VERY anti-catabolic. It also dopaminergic. Giving you the
benefits of increased CNS strength modulation by
its androgenic mode of action.
Androgens, in case you don't know, increase neuro-muscular
function, thus STRENGTH.
OK. Now, lets delve into the metabolic chemistry behind
dianabol's choice as a bridging agent.
When are testosterone levels highest?
Answer: In the AM, thats when.
Your body releases a tesosterone spike in the morning.
This is when tesosterone levels are highest.
When are Insulin levels lowest?
Answer: In the AM thats when.
Low insulin levels=increased protein used as fuel.
(Also fat, but protein is also being converted
to glucose via glucogenesis)
OK, here is where dball's short half-life works for us
(Its 3.2-4.5 hrs btw)
Lets take Subject X.
He's in bridging mode.
He has just woken up.
The body is about to release tesosterone, thus
creating a spike.
His insulin levels are low.
His LH and test levels are very low.
He pops 10mgs of dianabol.
Here is where things get interesting.
The 10mgs of dianabol will cause a testosterone
spike WHICH COINCIDES WITH the testosterone
released ENDOGENEOUSLY in the AM by the testes.
The body will be partially fooled.
It will not entirely detect the increased levels of testosterone
(above the normal test sipke), thus LH function WILL
REMAIN only partially(Very little actually) suppressed.
In other words, he is "piggy-backing" an extra dose of testosterone on top of the endogeneously reduced one,
thus creating an "inflated" test spike.
Henceforth, LH levels WILL BE ALLOWED TO SLOWLY
RECOVER over time.
Also, dballs anti-catabolic effect will help curb protein-loss
in the morning from low insulogenic levels.
HOWEVER, and here is where almost all of you go wrong.
You CANNOT GO PAST 10mg of dianabol in the AM
for this bridge to work!!!!
Why? Because of the blood levels of dianabol you would generate.
10mg in the AM will be broken down to 5mg in about 4 hrs
(Probably less)
5mg of dianabol, is not enough to cause another rise
in testosterone levels after the precceeding one. Thus,
LH function is allowed to up-regulate.
Anything more(Say 20mgs), will cause a SEDCONDARY
testosterone spike which WILL inhibit LH function further,
thus not allowing LH function to recover.
Oh yeah...100mgs? ROTLMFAO!! Fat chance.
The difference between 20mgs and 10mgs means the difference
between allowing LH to recover slowly and not allowing it to.
So, here's the scenario summed up:
Beginning: LOW LH and test.
Adding the 10mgs dball.
LH is allowed to SLOWLY RECOVER over time as
testosterone levels are kept at a level which
will not cause muscle-loss. Also, dball's anti-catabolic effects
will reduce protein degradation.(Via cortisone
reduction)
This is what i call a double positive. You have managed to
INCREASE anabolism(Test levels) and DECREASE
catabolism(cortisone), during a bridge to boot!!
The bridge should last 8 weeks, NO LESS.
I also have to say, that it WILL NOT restore
complete LH function. It'll get you 80-90%
of the way there but the only way you're going
to get your full LH function back is if you go OFF
completely.
Anavar WILL NOT restore LH completely either btw.
(In case anybody is wondering.)
The difference is that with anavar you can take it
throughout the day and with dball it HAS TO BE
once in the AM.
Hope that clears the air.
Fonz
From: http://boards.elitefitness.com/forum/showthread.php?threadid=150876
I've been reading some of the posts regarding this
bridge and some of them are truly from left-field.
First of, this is a BRIDGE. OK? a B-R-I-D-G-E.
Your LH function and Test levels are supposed
to RECOVER.
Ok, now having said that.
Here's the pharmo-kinetics behind Methandrostenelone,
brand name Dianabol.
10mg taken at once will increase your average testosterone level by 5 times and decrease your endogeneous cosrtisone
by 50-70%.
The reason why dianabol is a good choice for a bridge is that
its VERY anti-catabolic. It also dopaminergic. Giving you the
benefits of increased CNS strength modulation by
its androgenic mode of action.
Androgens, in case you don't know, increase neuro-muscular
function, thus STRENGTH.
OK. Now, lets delve into the metabolic chemistry behind
dianabol's choice as a bridging agent.
When are testosterone levels highest?
Answer: In the AM, thats when.
Your body releases a tesosterone spike in the morning.
This is when tesosterone levels are highest.
When are Insulin levels lowest?
Answer: In the AM thats when.
Low insulin levels=increased protein used as fuel.
(Also fat, but protein is also being converted
to glucose via glucogenesis)
OK, here is where dball's short half-life works for us
(Its 3.2-4.5 hrs btw)
Lets take Subject X.
He's in bridging mode.
He has just woken up.
The body is about to release tesosterone, thus
creating a spike.
His insulin levels are low.
His LH and test levels are very low.
He pops 10mgs of dianabol.
Here is where things get interesting.
The 10mgs of dianabol will cause a testosterone
spike WHICH COINCIDES WITH the testosterone
released ENDOGENEOUSLY in the AM by the testes.
The body will be partially fooled.
It will not entirely detect the increased levels of testosterone
(above the normal test sipke), thus LH function WILL
REMAIN only partially(Very little actually) suppressed.
In other words, he is "piggy-backing" an extra dose of testosterone on top of the endogeneously reduced one,
thus creating an "inflated" test spike.
Henceforth, LH levels WILL BE ALLOWED TO SLOWLY
RECOVER over time.
Also, dballs anti-catabolic effect will help curb protein-loss
in the morning from low insulogenic levels.
HOWEVER, and here is where almost all of you go wrong.
You CANNOT GO PAST 10mg of dianabol in the AM
for this bridge to work!!!!
Why? Because of the blood levels of dianabol you would generate.
10mg in the AM will be broken down to 5mg in about 4 hrs
(Probably less)
5mg of dianabol, is not enough to cause another rise
in testosterone levels after the precceeding one. Thus,
LH function is allowed to up-regulate.
Anything more(Say 20mgs), will cause a SEDCONDARY
testosterone spike which WILL inhibit LH function further,
thus not allowing LH function to recover.
Oh yeah...100mgs? ROTLMFAO!! Fat chance.
The difference between 20mgs and 10mgs means the difference
between allowing LH to recover slowly and not allowing it to.
So, here's the scenario summed up:
Beginning: LOW LH and test.
Adding the 10mgs dball.
LH is allowed to SLOWLY RECOVER over time as
testosterone levels are kept at a level which
will not cause muscle-loss. Also, dball's anti-catabolic effects
will reduce protein degradation.(Via cortisone
reduction)
This is what i call a double positive. You have managed to
INCREASE anabolism(Test levels) and DECREASE
catabolism(cortisone), during a bridge to boot!!
The bridge should last 8 weeks, NO LESS.
I also have to say, that it WILL NOT restore
complete LH function. It'll get you 80-90%
of the way there but the only way you're going
to get your full LH function back is if you go OFF
completely.
Anavar WILL NOT restore LH completely either btw.
(In case anybody is wondering.)
The difference is that with anavar you can take it
throughout the day and with dball it HAS TO BE
once in the AM.
Hope that clears the air.
Fonz
From: http://boards.elitefitness.com/forum/showthread.php?threadid=150876
DeepZenPill
New member
Heh. I must be slow tonight, I didn't even think of reposting it.
psychedout
New member
Thanks Rio 
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