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Does bromo really need to be split up during the day??
- Thread starter mvmaxx
- Start date
macrophage69alpha
New member
no it does not need to be split..
karma for the all knowing 
WannaImpress
New member
mvmaxx
are you using it for prolactin or for dieting?
are you using it for prolactin or for dieting?
WannaImpress
New member
barnidge said:
I've seen a few posts on here claiming that is has fat burning properties, so I'm curious if mvmaxx is using it for this reason!
WannaImpress said:
After reading johnboy's thread about it not really kicking in for 2-3 weeks I've decided to start it now before I start my prop/tren/winny cycle. I know I'm gyno prone and want to do everything in my power to stop from getting fina gyno.
So I will be starting Bromo 2 weeks before and I think I'll taper my way up from 1.25 to 2.5-5mg. I may keep it at 2.5mg until I feel the need to bump it to 5mg. We will see how I react to the sides. I also plan on starting Aromasin @ 25mg/ED 2 weeks prior as well.
Here's a post by Fonz regarding fat loss and bromo:
Bromocriptine works through dopaminergic pathways.
There are two ways to raget fa-loss via the ADRENERGIC
system.
1. The CNS is composed of the PNS and SNS.
PNS=Para-sympathetic nervous system
SNS=Sympathetic Nervous system
The SNS is governed by Adrenaline and Nor-adrenaline
The PNS is governed by DOPAMINE.
By increasing either Dopaime, adrenaline or nor-adrenaline,
you get an INCREASE in FFA breakdown.
Ephedrine, Clen etc.. stimulate the SNS.
Bromocriptine, DMAE, stimulate the PNS.
Having said all this, the use of PNS and SNS agonists
concurrently MAY overload the CNS of some people.
Result: Headches, migraines, twitching etc...
Either run one or the other.
What I do is switch back and forth between the 2.
W1-8: NYC+yohimburn W9-17: Bromocriptine
This way each adrenergic pathway is allowed to recover.
Fonz
Bromocriptine works through dopaminergic pathways.
There are two ways to raget fa-loss via the ADRENERGIC
system.
1. The CNS is composed of the PNS and SNS.
PNS=Para-sympathetic nervous system
SNS=Sympathetic Nervous system
The SNS is governed by Adrenaline and Nor-adrenaline
The PNS is governed by DOPAMINE.
By increasing either Dopaime, adrenaline or nor-adrenaline,
you get an INCREASE in FFA breakdown.
Ephedrine, Clen etc.. stimulate the SNS.
Bromocriptine, DMAE, stimulate the PNS.
Having said all this, the use of PNS and SNS agonists
concurrently MAY overload the CNS of some people.
Result: Headches, migraines, twitching etc...
Either run one or the other.
What I do is switch back and forth between the 2.
W1-8: NYC+yohimburn W9-17: Bromocriptine
This way each adrenergic pathway is allowed to recover.
Fonz
Bromocriptine
Bromocriptine is a dopamine agonist the has been shown to decrease body fat stores in experimental animals with little or no reduction in body weight or food intake.145 Accordingly, Meier and colleagues treated 33 obese postmenopausal women and 15 men with poorly-controlled Type II diabetes mellitus with bromocriptine. The subjects received 1.25 mg or 2.5 mg daily for 10 weeks with no other interventions. Body fat, as estimated by skinfold measurement, was reduced by 11.7% in the non-diabetic subjects. In the diabetic subjects, body fat was also reduced but to a greater extent in those subjects taking antidiabetic pills for control of their diabetes. Hyperglycemia resolved in most of the diabetics allowing cessation of hypoglycemic drugs in some. The degree of weight loss was minimal in all patients and mild nausea was the only side effect reported. 146 Encouraged by these results, the same investigators randomized 17 obese subjects to bromocriptine (1.6 or 2.4 mg daily within 2 hours of awakening of a quick-release form of bromocriptine) or placebo. In addition, all subjects were instructed to follow a moderately-low calorie diet. After 18 weeks, the subjects receiving bromocriptine had lost 6.3 kg and those receiving placebo only lost about 1 kg. Treatment with bromocriptine was also associated with a greater reduction in body fat and improved glucose tolerance.147 The way in which bromocriptine decreases fat stores and body weight and improves glucose tolerance is not known, but appetite suppression or a reduction of lipogenesis (the production of fat) have been proposed 146,147 Although these data are promising, the use of bromocriptine cannot be recommended until more information about long-term safety and efficacy is available. Bromocriptine is not currently FDA- approved for the treatment of obesity, but phase III studies are under way.
Also, here's a link:
http://www.ispub.com/journals/IJAPA/Vol1N2/obesity2.html
Bromocriptine is a dopamine agonist the has been shown to decrease body fat stores in experimental animals with little or no reduction in body weight or food intake.145 Accordingly, Meier and colleagues treated 33 obese postmenopausal women and 15 men with poorly-controlled Type II diabetes mellitus with bromocriptine. The subjects received 1.25 mg or 2.5 mg daily for 10 weeks with no other interventions. Body fat, as estimated by skinfold measurement, was reduced by 11.7% in the non-diabetic subjects. In the diabetic subjects, body fat was also reduced but to a greater extent in those subjects taking antidiabetic pills for control of their diabetes. Hyperglycemia resolved in most of the diabetics allowing cessation of hypoglycemic drugs in some. The degree of weight loss was minimal in all patients and mild nausea was the only side effect reported. 146 Encouraged by these results, the same investigators randomized 17 obese subjects to bromocriptine (1.6 or 2.4 mg daily within 2 hours of awakening of a quick-release form of bromocriptine) or placebo. In addition, all subjects were instructed to follow a moderately-low calorie diet. After 18 weeks, the subjects receiving bromocriptine had lost 6.3 kg and those receiving placebo only lost about 1 kg. Treatment with bromocriptine was also associated with a greater reduction in body fat and improved glucose tolerance.147 The way in which bromocriptine decreases fat stores and body weight and improves glucose tolerance is not known, but appetite suppression or a reduction of lipogenesis (the production of fat) have been proposed 146,147 Although these data are promising, the use of bromocriptine cannot be recommended until more information about long-term safety and efficacy is available. Bromocriptine is not currently FDA- approved for the treatment of obesity, but phase III studies are under way.
Also, here's a link:
http://www.ispub.com/journals/IJAPA/Vol1N2/obesity2.html
PopiChulo said:
I will be starting at 1.25mg/ED for a week and will then go to 2.5mg/ED. I'll take 2.5mg at night before bed. If the sides get too bad I'll split them up. But I'm hoping I get past the first week a little easier since I'm doing a low dose.
BTW, you owe me an e-mail.
MrQuestion
Banned
Take the bromo as soon as you wake up in the day. YOu are wasting it taking it at night.
MrQuestion
Banned
mvmaxx said:
Circadiam Rythms (yes, I misspelled that word), dopamine being very low in the day time yet higher at night. There are a plenty of studies that show this.
Someone else can explain it better than I can. Im too lazy to go into detail at the moment.
MrQuestion said:
Actually, splitting the dose works best.
1. You get a dopamine increase during the day and:
2.YOU GET THE ADDED BENEFITS OF INCREASED GH SECRETION
BECAUSE OF PROLACTIN INHIBITION during the day AND night.
If you only took it in the AM, your night-time GH releaase
would be lower than if you took the Bromo in the AM and PM.
Best dose, that people TOLERATE is 1.25mgs in the AM and PM
(For a total of 2.5mg).
2.5mgs in the AM and PM is also doable(For a total of 5mg), but it will take some time to ramp up to this level.
Fonz
SuperWoman
New member
Bromo's half life is 10hrs. It reaches peak concentration in your blood within 2-3 hrs after taking it. No studies show that breaking up the dosage alter results with fat loss. Do not take it at night unless you want your sides to be worse. Take in the AM with food. Do not take with ephedrine or clen unless you want to waste your bromo as these compounds block bromo's effects.
read lyle mcdonald's book at qfac.com it explains everything you ever wanted to know about bromo for dieting, post roid cycle, to loose fat and gain muscle.
read lyle mcdonald's book at qfac.com it explains everything you ever wanted to know about bromo for dieting, post roid cycle, to loose fat and gain muscle.
S
Spartacus
Guest
what would happen if you mixed 200mg of wellbutrin sr daily with some bromo?..I cna't find any contraindications within the rather lengthy prescribing info insert..as you may know wellbutrin/bupropion is an inhibitor of the neuronal uptake of norepinephrine and dopamine..and therefore its mechanisms of action are mediated by noradrenergic and/or dopaminergic mechanisms..would stacking bromo with wellbutrin be a possible scenario for overload?...I've just started wellbutrin a week ago and I love it..my lifts are more intense(w/o any thermogenics etc)and my appetite is much more manageable
S
Spartacus
Guest
bump
S
Spartacus
Guest
I took my first 2.5mg last night and I had no appetite..and usually I have a voracious appetite at night after a work-out..with this stuff I quit eating a bowl of bran cereal mid-way thru as I just wasn't hungry
Fonz, can you explain the reasoning behind taking Octopmaine with the bromo. Can you also suggest a dosage regimen.
BTW, I found a link for it if anyone is interested.
http://www.affordablesupplements.com/beta3.asp
BTW, I found a link for it if anyone is interested.
http://www.affordablesupplements.com/beta3.asp
mvmaxx said:
Octopamine does not cause an adrenaline like other beta agonists.
Its the adrenaline surge that cancels out the dopamine
surge caused by the Bromocriptine.
Octopamine only stimulates BAT(Brown Adipose Tissue),
causing a rise in your temp. due to muscular shivering).
Fonz
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