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Clomid vs Nolvadex
- Thread starter BIGRICH
- Start date
YUP can be used in the same manner as clomid. Takes longer but works. In fact Nolva brought my test up alot better then clomid believe it or not...go figure
If you can't get clomid I would use it...My doc had me on clomid for a while then switched me over becasue the clomid was not working. I got a better reaction with the nolva
He is going to give me armidex next
If you can't get clomid I would use it...My doc had me on clomid for a while then switched me over becasue the clomid was not working. I got a better reaction with the nolva
He is going to give me armidex next
Both Clomid and Nolvadex are used in treating male infertility
Both clomiphene citrate and tamoxifen are estrogen receptor blockers that have been suggested as empiric treatments for male infertility. By preventing the important negative feedback of estrogens to the pituitary and hypothalamus, LH/FSH pulsatile release and GnRH stimuli are augmented. Since FSH is important for spermatogenesis, it is possible that increased FSH release may further enhance sperm production. Increased LH release also results in higher serum testosterone levels that are converted peripherally as well as in the liver to estrogens. Since men with idiopathic infertility have normal testosterone levels, by definition, the increased FSH, LH and testosterone that result from clomiphene or tamoxifen treatment may boost testosterone and estrogen levels above normal levels. This increased estrogen production may be detrimental to normal sperm production and should be avoided. Therefore, all patients considered for empiric therapy should be counselled to have early and frequent testosterone and estradiol levels to monitor treatment.
Aromatase inhibitors block the conversion of testosterone to estrogen. Treatment with an aromatase inhibitor decreases estrogen levels, which leads to increased LH and FSH release from the pituitary, with a subsequent increase in testicular stimulation and serum testosterone levels without the usual increase in estrogen levels seen for anti-estrogens. Although one uncontrolled study suggested an increase in sperm concentration for nearly all patients treated (89%), other well-designed placebo-controlled studies have demonstrated no significant improvement in pregnancy rates for treated patients. Although many patients will have increased sperm concentration, no improvement in sperm motility was seen in these studies.
http://www.maleinfertility.org/new-therapy.html#anti
Both clomiphene citrate and tamoxifen are estrogen receptor blockers that have been suggested as empiric treatments for male infertility. By preventing the important negative feedback of estrogens to the pituitary and hypothalamus, LH/FSH pulsatile release and GnRH stimuli are augmented. Since FSH is important for spermatogenesis, it is possible that increased FSH release may further enhance sperm production. Increased LH release also results in higher serum testosterone levels that are converted peripherally as well as in the liver to estrogens. Since men with idiopathic infertility have normal testosterone levels, by definition, the increased FSH, LH and testosterone that result from clomiphene or tamoxifen treatment may boost testosterone and estrogen levels above normal levels. This increased estrogen production may be detrimental to normal sperm production and should be avoided. Therefore, all patients considered for empiric therapy should be counselled to have early and frequent testosterone and estradiol levels to monitor treatment.
Aromatase inhibitors block the conversion of testosterone to estrogen. Treatment with an aromatase inhibitor decreases estrogen levels, which leads to increased LH and FSH release from the pituitary, with a subsequent increase in testicular stimulation and serum testosterone levels without the usual increase in estrogen levels seen for anti-estrogens. Although one uncontrolled study suggested an increase in sperm concentration for nearly all patients treated (89%), other well-designed placebo-controlled studies have demonstrated no significant improvement in pregnancy rates for treated patients. Although many patients will have increased sperm concentration, no improvement in sperm motility was seen in these studies.
http://www.maleinfertility.org/new-therapy.html#anti
macrophage69alpha
New member
clomifene and tamoxifin DO NOT HAVE THE SAME ACTIVITY IN THE SAME TISSUES. There is considerable differences in their activity in breast tissue (tamoxifin has GREATER affinity here) and in the anterior pituitary(clomifene has greater affinity here).
they also have different affinity in uterus...
peace
they also have different affinity in uterus...
peace
Can we end once and for all the confusion with the essential drugs for steroid using bodybuilders i.e. anti-estrogens?
What kind of thing am I looking for?
What proof exists that clomid is better at blocking estrogen at the pituitary?????????????
If Nolvadex is 7 times stronger than clomid on a per mg basis (based on an argument by Llewellyn) then surely Nolvadex is the better choice?
Clomid seemed to do fuck all for me following my cycle. I will not be using clomid next time. I will use Proviron and Nolvadex instead.
Anyoone? Discuss?
What kind of thing am I looking for?
What proof exists that clomid is better at blocking estrogen at the pituitary?????????????
If Nolvadex is 7 times stronger than clomid on a per mg basis (based on an argument by Llewellyn) then surely Nolvadex is the better choice?
Clomid seemed to do fuck all for me following my cycle. I will not be using clomid next time. I will use Proviron and Nolvadex instead.
Anyoone? Discuss?
sins its a androgen....... some say it dos some say no chicho1234
New member
I cant get any clomid here right now and I have a shitload of noveldex right now!!!. I am almost done sustanon 250 a week, how much noveldex should I use post cycle and how long?? THanks guys!!!!!!!!!!!
