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Class I vs. Class II: What's What?

:)

Anabolic Steroids achieve their effects on hypertrophy through different mechanisms,and as such are classified into two different categories,class-I and class-II compounds.I will explain which ones work through which pathway,and how to effectively combine opposing groups to maximize potentiation of one another for explosive growth....

Class-I-These are steroids who's primary influence on anabolism is achieved through aggressive binding and activation of the androgen receptor...Examples of potent class-I's are-Deca-durabolin,primobolan,equipoise,oxandrolone...

Class-II-These are compounds with potent activity independent of A/R binding/activation,and their activity has been monitored in neuron's,microsomes,mitochondria,etc...Examples of potent class-II's are-Anadrol,Dianabol,winstrol,Fluoxymesterone...

Then we have steroids that are potent combination steroids all by themselves(meaning they display influences on growth through both class-I and II activity,and thus are very effective as'stand-alone'anabolic agents as well as forming the 'base' of most steroid stacks)....Two compounds possess this unique characteristic-Trenbelone and Testosterone.Either of these two steroids should form the base of most users stacks,as the cover both A/R and non-A/R mediated mechanisms,and adding to them with either a class-I or II will only potentiate that particular mechanism towards muscular hypertrophy...

Now that we're aware of which compounds work through which mechanism,how do we combine them effectively to maximize each one's potential?As explained above,Testosterone and trenbelone should form the base of any serious steroid stack,but great effects can be had by combining single mechanism steroids from opposing classes....Examples of this type of synergistic combining could be(but are not limited to)

*CUTTING*
Winstrol & Equipoise
Winstrol & Primobolan
Fluoxymesterone & Equipoise or Primobolan

*BULKING*
Deca & Dianabol
Anadrol & Equipoise or Primobolan
Winstrol & deca

And of course,using test or tren as the base compound for either cutting or bulking will only make results that much more explosive.If using test for cutting,an anti-aromitase should be incorporated...
 
????? If you are asking where they fit on the Controlled Substances List, then its CIII(3). Thats what I was assuming you were asking about.
 
I think alot of the recent abstracts posted on this board (i.e, tren hitting different receptor, nor derivatives increasing IGF1, anavar acting on phosphocreatine synthase, etc. really show that simply catagorizing a steroid as Type I or Type II is a very big over-generalization.... it is like catagorizing all women into either "blonds" or "brunettes." I mean we have some serious distinctive features within the classes themselves. For example a particular blond might stand out because she has large hooters or gives a killer hummer, just as a particular "Type I" might stand out for its ability to lubricate joints, promote hunger, etc.

Bill Roberts is not god...you have to remember that. If you look at his catagories they really fall into injectables vs. 17aa orals. 17aa drugs are different because they do seem to have the ability to stimulate growth factors in the liver and have a strong anti-catabolic effect. So yes, stacking an injectable (which can be dosed high because it is less hepatoxic) and an oral is a good idea. You don't have to buy into this "type I, II, IIA xyz" mental masturbation. Anyway, half the reason of stacking is not synergy, but getting enough mgs of steroid into your body without too many side effects. For example, while eq and deca might in some ways perform the same task (personally I think they have very unique differences though) using 300mg deca a week and 300mg eq a week might very well be a better choice than just 600mg deca a week...why? At 600 mg the deca is much more likely to cause gyno issues. Same with type IIs... 50mg of winstrol a day will give growth, but also could likely give joint pain. 25mg of winstrol a day + 25mg of some other oral would be a better choice in avoiding this side effect while still getting 50mg a day into your system.

The type I and II classification also ignores the fact that steroids can convert to different subtrates in the body. For example when you shoot test you are not just raising test levels...you will end up raising estrogen and DHT levels. Alot of the benifits from test are from estrogen causing increased water retention (better leverage) and the DHT causing aggression (use test with proscar and you will realize what I am saying. Even on a gram of test, preventing DHT conversion really lowers sex drive and the kill the weights feeling you get from just test). I mean come on, there are hundreds of other variables that effect the results of steroid therapy, not just two.
 
heh.........

i remember when this thread arose.....a classic.

my .02 here:....lol

ive read the literature and it seems that oxandrolone is unique among oral 17-AA steroids, as the majority of orals are Class II steroids giving most of their anabolic effect by means other than the androgen receptor (AR).

oxandrolone shows a good binding to the AR.........is it therefore to be considered more a Class I steroid?

so my query is would one benifit from stacking oxandrolone along with a Class II steroid? if so, what would be a synergistic combo?

Unity66
 
Yes,ox would stack well with just about any other A/S,and HG makes a very good point,the classing of anabolics into just 2 categories doesn't do them proper justice,as even the steroids which roughly fall into the same category will illicite very differing responses from one another/from individual to individual...Take as an example,primo and deca,these are both tossed into the Class-I category(as both are almost PRIMARILY A/R binders),yet most users will agree that deca produces much stronger effects than primo...So if they both work by the same basic mechanism,why the difference?The answer is multi-faceted.Deca's binding capacity to the A/R is much more aggressive than primo,not to mention it exhibits some progestogenic activity that primo does not...I would hasten a guess that when deca interacts with DNA the signals it sends out are slightly differing in one degree or another from primo,and the same scenario can apply to all other compounds,as well as everyone's slightly differing genetic responses to various signals...
 
Damn, Huck. How much does elite pay you for this.

Just a word of appreciation for your dedication to knowledge and your willingness to help others.
 
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