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What's the difference between test alone and test with Finasteride or Dutasteride?
(From those that have tried them both)
That is,
test and dht via 5AR
-vs-
test and no or reduced dht via 5AR inhibition (finas or dutas)
I've always relied on topical 5AR inhibition (via azelaic acid or spironolactone) rather than systemic 5AR inhibition (finasteride or dutasteride).
I'm assuming there must be a HUGE difference of test without systemic conversion to DHT (a much more potent androgen).
What is that difference in terms of real world experience (empirical evidence)?
(From those that have tried them both)
That is,
test and dht via 5AR
-vs-
test and no or reduced dht via 5AR inhibition (finas or dutas)
I've always relied on topical 5AR inhibition (via azelaic acid or spironolactone) rather than systemic 5AR inhibition (finasteride or dutasteride).
I'm assuming there must be a HUGE difference of test without systemic conversion to DHT (a much more potent androgen).
What is that difference in terms of real world experience (empirical evidence)?

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