Yeah, thats the sad, an ounce of prevention type of thing. Same with chest pain, I had two patients to assess the same day, both had heart attacks with near identical blockages in the left anterior descending artery the major vessel feeding the LV and both similar in age in their forties. One came in immediately, the other waited a few hours, the one who came in LV heart function was near normal with minimal amount of stunned muscle or hibernating, hardly tell. The one who waited the LV function was severely diminished, mortality and morbity of the second skyrocketing just b/c they waited. Chest pain is not anything to mess with. Same with bp, the number one reason people have congestive heart failure. And so preventable.
The heart gets all baggy. We get the thrombus in apex of the heart/left ventricle usually from a heart attack though, b/c of LV shape and apex is fed by the distal LAD and so any blockage along its length knocks out the apex. Conversely in the CHF patients or atrial fib patients its the atrium clots we see. Ive seen some whoppers. Mainly cuz the ventricle still moves blood forcefully. Atrium however gets baggy and loses contractility sooner, the blood just gets stagnant in there, forms a thrombus on the back wall usually just waiting to break off and shoot up to the brain. So we give them coumadin which does the opposite thing dissolves the clot, but if they have a brain bleed a different form of stroke, its more severe. Quite a pickle at times and unnecessary.CHF is chiefly why they came up with the new lower BP guidelines in 2003.