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Just a few things I have been thinking about lately, so bear with me…
Permanent gains…hmmm, various and sundry posters cry out that steroid gains aren’t permanent. This reminds me of my grandmother when she told me a few years ago that dieting doesn’t work. I asked her what she meant. And she told me that she dieted once about 20 years ago, and look at her now all fat. ‘Obviously,’ she said, ‘dieting doesn’t work.’ The point is that your body is always in flux. It’s growing, shrinking, anabolic, catabolic, etc., etc. You can bitch about it, or you can persevere. The choice is yours.
Short cycles…this just seems like a piss poor idea to me. Here is my thought process on them: the majority of initial gains aren’t contractile tissue. Take oxandrolone for example, a drug that most will tell you the weight it adds is ‘permanent’ (there’s that term again, lol). Well, one of the things it does is allow the muscle to store more creatine than normal. Remember how creatine got to be so popular? Guys were gaining 10 pounds of ‘lean mass’ in a week or so, that’s how. Because creatine allows you to hold more water in your muscle, yadda yadda yadda. My point is the beginning of your cycle the gear is changing the environment of your body to quickly gain actual contractile tissue. This is the LAST thing you gain, after all the infrastructure has been put into place by the gear. And there really is no telling what all that entails. Anyways, the initial process takes time. Sure, you make ‘quick’ gains the first 4-6 weeks, after that, not so quick. Why is that? Because actually adding muscle ain’t fucking easy. Ever wonder why guys who say they are going to do those short cycles always tell you about the first cycle, but never any others? I don’t wonder.
Staying on ‘too long’… is that possible? I mean really? And how do you know? ‘Oh, Mr. Tx, I know because if you stay on for longer than 16 weeks it’s harder to get your hpta back up and running.’ Ok. And what happens when we age? Our test levels fall like a rock. And how high were your levels beforehand? What is hormone replacement therapy? One looooooong ass low dose cycle. Don’t get me wrong, you should be aware that if you do this on your own there are obviously risks involved. Consult with your doctor.
Ttokkyo primo…what the fuck is up with that? It’s like Geraldo Rivera with Al Capone’s vault, I feel like breaking somebody’s legs already. Capiche?
Lot’s of gyno questions being asked of me lately…I have come to the conclusion that most of the gyno problems from deca, fina, and anadrol can be stopped now and EVEN REVERSED if acted upon in time. Of course, it may be more cost-effective to go with other compounds because of the expense of the solution. Anyways, there are 4 ways we are aware of at this time to get gyno and my solutions for these guys:
progestin based – you are fucked, but you can always hope for ru-486
prolactin based – vitex at a gram/day to begin…maintenance ~half that
igf-1 based – nolvadex reduces igf ~58%, clomid ~30%, arimidex ~18%
estrogen rebound – nolvadex, anastrozole, or the new one letrozole (?)
And remember, it can be a combo of all of the above, so be careful. I think if you find out you get gyno symptoms from these compounds (and not until that happens) then you should hit nolva/vitex combo. Like 10mg/day of nolva and 500mg-gram/day of vitex. You will have to play with this to see what you respond to. Feel free to experiment.
And for those who made it all the way through, you’ve got way too much time on your hands, lol.
Permanent gains…hmmm, various and sundry posters cry out that steroid gains aren’t permanent. This reminds me of my grandmother when she told me a few years ago that dieting doesn’t work. I asked her what she meant. And she told me that she dieted once about 20 years ago, and look at her now all fat. ‘Obviously,’ she said, ‘dieting doesn’t work.’ The point is that your body is always in flux. It’s growing, shrinking, anabolic, catabolic, etc., etc. You can bitch about it, or you can persevere. The choice is yours.
Short cycles…this just seems like a piss poor idea to me. Here is my thought process on them: the majority of initial gains aren’t contractile tissue. Take oxandrolone for example, a drug that most will tell you the weight it adds is ‘permanent’ (there’s that term again, lol). Well, one of the things it does is allow the muscle to store more creatine than normal. Remember how creatine got to be so popular? Guys were gaining 10 pounds of ‘lean mass’ in a week or so, that’s how. Because creatine allows you to hold more water in your muscle, yadda yadda yadda. My point is the beginning of your cycle the gear is changing the environment of your body to quickly gain actual contractile tissue. This is the LAST thing you gain, after all the infrastructure has been put into place by the gear. And there really is no telling what all that entails. Anyways, the initial process takes time. Sure, you make ‘quick’ gains the first 4-6 weeks, after that, not so quick. Why is that? Because actually adding muscle ain’t fucking easy. Ever wonder why guys who say they are going to do those short cycles always tell you about the first cycle, but never any others? I don’t wonder.
Staying on ‘too long’… is that possible? I mean really? And how do you know? ‘Oh, Mr. Tx, I know because if you stay on for longer than 16 weeks it’s harder to get your hpta back up and running.’ Ok. And what happens when we age? Our test levels fall like a rock. And how high were your levels beforehand? What is hormone replacement therapy? One looooooong ass low dose cycle. Don’t get me wrong, you should be aware that if you do this on your own there are obviously risks involved. Consult with your doctor.
Ttokkyo primo…what the fuck is up with that? It’s like Geraldo Rivera with Al Capone’s vault, I feel like breaking somebody’s legs already. Capiche?
Lot’s of gyno questions being asked of me lately…I have come to the conclusion that most of the gyno problems from deca, fina, and anadrol can be stopped now and EVEN REVERSED if acted upon in time. Of course, it may be more cost-effective to go with other compounds because of the expense of the solution. Anyways, there are 4 ways we are aware of at this time to get gyno and my solutions for these guys:
progestin based – you are fucked, but you can always hope for ru-486
prolactin based – vitex at a gram/day to begin…maintenance ~half that
igf-1 based – nolvadex reduces igf ~58%, clomid ~30%, arimidex ~18%
estrogen rebound – nolvadex, anastrozole, or the new one letrozole (?)
And remember, it can be a combo of all of the above, so be careful. I think if you find out you get gyno symptoms from these compounds (and not until that happens) then you should hit nolva/vitex combo. Like 10mg/day of nolva and 500mg-gram/day of vitex. You will have to play with this to see what you respond to. Feel free to experiment.
And for those who made it all the way through, you’ve got way too much time on your hands, lol.
