You want me to do all the work......u could have explained
All you have to do is do a search......
you want to raise your nat T levels after a cycle since test at 400mg a week will shut down you LH production resulting in Low T levels. You want to get your T levels back up asap to retain gains
And you can use Nova to raise nat test levels, my endo has used it with me and it raised my T levels better then clomid.........different peoples bodies react to different ant e's differently
clomid is more potenet in most becasue it binds right to receptors in the pituitary where Nolva works through ( I think ) the breast receptors hence preventing gyno
Arimidex is another option to raise nat test this does not block it but actually eliminates it..do a search
Here is a good post cut and pasted explaining anti e's and raising nat t levels
Both clomiphene citrate and tamoxifen are estrogen receptor blockers that have been suggested as empiric treatments for male infertility. By preventing the important negative feedback of estrogens to the pituitary and hypothalamus, LH/FSH pulsatile release and GnRH stimuli are augmented. Since FSH is important for spermatogenesis, it is possible that increased FSH release may further enhance sperm production. Increased LH release also results in higher serum testosterone levels that are converted peripherally as well as in the liver to estrogens. Since men with idiopathic infertility have normal testosterone levels, by definition, the increased FSH, LH and testosterone that result from clomiphene or tamoxifen treatment may boost testosterone and estrogen levels above normal levels. This increased estrogen production may be detrimental to normal sperm production and should be avoided. Therefore, all patients considered for empiric therapy should be counselled to have early and frequent testosterone and estradiol levels to monitor treatment.
Aromatase inhibitors block the conversion of testosterone to estrogen. Treatment with an aromatase inhibitor decreases estrogen levels, which leads to increased LH and FSH release from the pituitary, with a subsequent increase in testicular stimulation and serum testosterone levels without the usual increase in estrogen levels seen for anti-estrogens. Although one uncontrolled study suggested an increase in sperm concentration for nearly all patients treated (89%), other well-designed placebo-controlled studies have demonstrated no significant improvement in pregnancy rates for treated patients. Although many patients will have increased sperm concentration, no improvement in sperm motility was seen in these studies.
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