No, it is like Deca in the sense that it may convert. Here's a great post by Animal that was just posted on my Fina board:
tren doesn't convert to E, we know that. However, within the last 2 weeks on one of these sites is a post that tren is progestenic in and bound better to certain progesterone receptors.
Well, I still had the files up, so those studies are below. BTW, it's been shown that deca can convert to estrogen and progesterone and not just hit those receptors.
You may think your hair is falling out, but it's being replaced just as fast. Look up tren in the merck
and then look up hirsuitism.
Yes, tren cause gyno and anyone that says it can't is an idiot. Gyno can and is caused by IGF-1
mediated affects in addition to estrogen. Gee, I wonder if that's why they use nolvadex which stops IGF-1 production?
Read it and weep because sure as shit it says that tren binds better to the PR than progesterone!
' For 17beta-trenbolone (17beta-TbOH), the active compound after TBA administration, an affinity the rhAR similar to dihydrotestosterone (DHT) and a slightly higher affinity to the bPR than progesterone were demonstrated.
Now who's knows what and the entire study is below.
Characterisation of the affinity of different anabolics and synthetic hormones to the human androgen
receptor, human sex hormone binding globulin and to the bovine progestin receptor.
APMIS 2000 Dec;108(12):838-46 (ISSN: 0903-4641)
Bauer ER; Daxenberger A; Petri T; Sauerwein H; Meyer HH [Find other articles with these Authors]
Institut fur Physiologie, Research Center for Milk and Food Weihenstephan, Technical University
Munich, Germany.
For the steroidal growth promoters trenbolone acetate (TBA) and melengestrol acetate (MGA) neither
the complete spectrum of biological activities nor the potential endocrine disrupting activity of their
excreted metabolites in the environment is fully understood. The potency of these substances in
[3H]dihydrotestosterone ([3H]-DHT) displacement from the recombinant human androgen receptor
(rhAR) and from human sex-hormone binding globulin (hSHBG) was evaluated. In addition, the
potency for [3H]-ORG2058 displacement from the bovine uterine progestin receptor (bPR) was tested.
For comparison, different anabolics and synthetic hormones were also tested for their binding
affinities. For 17beta-trenbolone (17beta-TbOH), the active compound after TBA administration, an
affinity the rhAR similar to dihydrotestosterone (DHT) and a slightly higher affinity to the bPR than
progesterone were demonstrated. The affinity of the two major metabolites, 17alpha-trenbolone and
trendione, was reduced to less than 5% of the 17beta-TbOH-value. The affinity of these three
compounds and of MGA to the hSHBG was much lower compared with DHT. MGA showed a 5.3-fold
higher affinity than progesterone to the bPR but only a weak affinity to the rhAR. The major MGA
metabolites have an affinity to the bPR between 85% and 28% of the affinity of progesterone. In
consequence, MGA and TBA metabolites may be hormonally active substances, which will be present
in edible tissues and in manure. We conclude that detailed investigations on biodegradation,
distribution and bio-efficacy of these substances are necessary.
Replied By
Small
Ultimate Power
USA
468 Posts
Posted - May 02 2001 : 21:08:33
Environ Qual Saf Suppl 1976;(5):253-64 Related Articles, Books, LinkOut
Pharmacological and endocrinological studies on anabolic agents.
Neumann F
When used in connection with animal production the term "anabolic agents" covers a wide range. Ther
steroidal male and female sex hormones are included in this list, as are the nonsteroidal estrogens.
For the clinician and for the endocrinologist, anabolics are only @#%$ chemically related to
testosterone and 19-nortestosterone. Estrogens, though possessing anabolic properties, too, do not
belong to this class. This paper will deal with anabolic agents in in the stricter sense of which mainly
trenbolone acetate combined with hexestrol has been recommended for bull and heifer fattening. To
consider possible consumer injury from ingestion of meat from anabolic agent treated animals, it is
necessary to know the pharmacological properties of the agents, the doses producing certain effects
or might produce, and the levels of residues in the meat. Trenbolone acetate will be compared with
the following anabolic agents: methenolone acetate, methandrostenolone, nandrone, androstanazole,
and 19-nortestosterone. The activity spectrum of trenbolone acetate is similar to that of
19-nortestosterone or those anabolics that are derived from 19-nortestosterone. The compound has
about three times stronger androgenic effect than testosterone propionate. Its index of dissociation
between anabolic/androgenic activity is 2--3. This index is 3--10 for the other anabolic agents. As
regards the virilizing potency, trenbolone acetate is also on the top of the list. It seems that
androgenicity and degree of virilization run paralle. The antigonadotropic activity (inhibition of
ovulation and testicular growth) of trenbolone acetate exceeds that of testosterone propionate by the
factor 3.
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