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The C-terminus of the HGH molecule has been isolated and it has been determined to be responsible for the specific effect on fat. It stimulates lipase (enzyme responsible for lipolysis) , inhibits acetyl-CoA carboxylase (enzyme responsible for fat storage) and it may have an effect on Beta-3 AR's. This strand is being developed into a medication and it is expected to be out in four years. The problem with using rHGH as an advanced obesity drug is the insulin resistence, organ/tissue growth (hyperplasia), and hypothyroidism. This new med will not cause any of these side effects. Of course, these sides can be eliminated if the user uses a low carb diet to combat the insulin resistence, uses Armour thyroid to overcome the hypothyroidism, and keeps the doses low enough to avoid unwanted tissue growth, joint pain, and swelling.
People are dropping 1.1 to 2.2 pounds per week with "normal eating habits." This will be an oral medication. So, some good news for the high BF%'s. Unfortunately, we will have to wait another four years.
People are dropping 1.1 to 2.2 pounds per week with "normal eating habits." This will be an oral medication. So, some good news for the high BF%'s. Unfortunately, we will have to wait another four years.
