fina side effects

[meat19]

besides cranberry extract what else can i use to protect my kidneys? does fina cause increased prolactin levels? how much chasteberry do i need to take to offset that. what does prolactin do for guys?

 

[wrj]

I believe prolactin is the natural anti GH that your body produces and I've heard that its a myth that fina raises prolactin levels. But I'm not sure on either.
As for your kidneys, just drink shit loads of water with your cranberry extract and you'll be fine.

Bump for more solid info.

WRJ

 

[Zyglamail]

I believe prolactin is the natural anti GH that your body produces and I've heard that its a myth that fina raises prolactin levels

Human studies with tren are very limited and animal studies dont often look for prolactin so the evedence is weak. On a side note however, there are guys here who have, shortly after fina cycle have had blood work done and have had elevated prolactine levels. I will see what I can dig up.

 

[freshr1]

Human studies with tren are very limited and animal studies dont often look for prolactin so the evedence is weak. On a side note however, there are guys here who have, shortly after fina cycle have had blood work done and have had elevated prolactine levels. I will see what I can dig up.

Shitty... keep us posted on this please! thanks.

 

[Zyglamail]

Ok, while direct studies showing the correlation are hard to find if you read these abstracts, you can likely draw a conclusion. The first Abstract here shows that tren causes an increase in igf-1. Granted, the weakness I find in many of the abstracts referre to tren and estradiol.

Growth and hormonal response of intact and castrate male cattle to trenbolone acetate and estradiol.

Lee CY, Henricks DM, Skelley GC, Grimes LW.

Dept. of Anim. Sci., Clemson University, SC 29634.

Effects of castration and anabolic implants on weight gain, rib soft tissue composition and serum hormones were studied in cattle using a completely random design with a 2 x 2 factorial arrangement. Half of 16 bulls and 16 steers (Angus or Angus x Brahman) aged 9 mo and weighing 290 kg were treated with an implant (200 mg trenbolone acetate and 24 mg estradiol). Half of each group were not treated with an implant. A growing diet was fed for 95 d and half the animals in each group were slaughtered. Animals in the treated groups were reimplanted with trenbolone acetate and fed a finishing diet for 84 d and slaughtered. Percentage dry matter, fat and protein were determined on soft tissue from the 9-10-11th rib. Two blood samples were collected from each animal every 2 wk. Serum was assayed for five hormones. During the growing phase, untreated and treated bulls and treated steers gained more weight and had leaner rib sections that untreated steers (P less than .05); after the finishing phase, there were no differences among groups. Untreated steers had lower insulin-like growth factor (IGF-I) and higher cortisol concentrations during both phases of growth than untreated bulls did (P less than .05). Treatment with implants increased IGF-I concentrations in steers during both phases and reduced cortisol during the finishing phase.(ABSTRACT TRUNCATED AT 250 WORDS)

And this one shows how IGF-1 is a stimulator for the release of prolactin.

Insulin-like growth factor-I augments prolactin and inhibits growth hormone release through distinct as well as overlapping cellular signaling pathways.

Fruchtman S, Gift B, Howes B, Borski R.

Department of Zoology, North Carolina State University, Box 7617, Raleigh, NC 27695-7617, USA.

We recently discovered a new role for insulin-like growth factor-I (IGF-I) as a specific and direct stimulator of prolactin (PRL) release in addition to its recognized function as an inhibitor of growth hormone (GH) release and synthesis. Little is known of the mechanisms that transduce the actions of IGF-I on PRL and GH release in vertebrates. The present study was undertaken to determine the cellular pathways that mediate the disparate actions of IGF-I on PRL and GH release in hybrid striped bass (Morone saxatilis X M. chrysops). When regulating cellular function, IGF-I may activate two primary pathways, phosphatidylinositol 3-kinase (PI 3-K) and mitogen-activated protein kinase (MAPK). The specific MAPK inhibitor, PD98059, blocked IGF-I-evoked PRL release as well as GH release inhibition over an 18-20-h incubation. LY294002, a specific PI 3-K inhibitor, overcame IGF-I's inhibition of GH release but was ineffective in blocking PRL release stimulated by IGF-I. These studies suggest IGF-I disparately alters PRL and GH by activating distinct as well as overlapping signaling pathways central for mediating actions of growth factors on secretory activity as well as cell proliferation. These results further support a role for IGF-I as a physiological regulator of PRL and GH.

 

[meat19]

will 500mg of chasteberry a day decrease prolactin while on fina?