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I just read the following article off www.anabolicreview.com it mentions the use of clenbuterol and Ephedrine as anticatabolics when coming off of a cycle. I believe this to be true for clenbuterol but what do you think about the use of Ephedrine as I have heard differing opinion
All this, however, helps only if the athlete is able to mostly block out the catabolic effect of the increased cortisone level. A compound which, because of its distinct anticatabolic effect, fulfills this requirement is the beta-2 sympathomimetic, Clenbuterol. Clenbuterol successfully blocks the cortisone receptors so that the athlete is usually able to maintain a large portion of the strength and muscle mass built up by the steroids. The intake of Clenbuterol begins directly at the end of the steroid therapy and continues over 8-10 weeks (see also Clenbuterol). Another compound of the group of sympaticomimetics which also has an anticatabolic effect (but less pronounced than Clenbuterol) is Ephedrine. Probably the most suitable drug in this situation is a preparation which in school medicine is used in the treatment of the Cushing's syndrome, a hyperfunction of the adrenal glands which causes the body to produce too much cortisone. Those who have read this book carefully will know which drug is meant: Cytadren. Since it reduces the cortisone level extremely well athletes usually take it directly after completion of a steroid treatment (see also Cytadren). Several athletes take thyroid hormones in this phase since they have an anabolic effect when taken in small dosages and for not excessively long intake intervals. Their effect can be clearly increased by the anticatabolic effect of Clenbuterol which explains why this combination is used during the phase of discontinuance. The use of growth hormone also makes sense since it has a strong anticatabolic/anabolic effect. You can forget Ornithin and Arginin which supposedly increase the realising of GH, because they are ineffective. Distance yourself from the thought that pharmaceutically improved muscle mass can be maintained with "natural methods."
All this, however, helps only if the athlete is able to mostly block out the catabolic effect of the increased cortisone level. A compound which, because of its distinct anticatabolic effect, fulfills this requirement is the beta-2 sympathomimetic, Clenbuterol. Clenbuterol successfully blocks the cortisone receptors so that the athlete is usually able to maintain a large portion of the strength and muscle mass built up by the steroids. The intake of Clenbuterol begins directly at the end of the steroid therapy and continues over 8-10 weeks (see also Clenbuterol). Another compound of the group of sympaticomimetics which also has an anticatabolic effect (but less pronounced than Clenbuterol) is Ephedrine. Probably the most suitable drug in this situation is a preparation which in school medicine is used in the treatment of the Cushing's syndrome, a hyperfunction of the adrenal glands which causes the body to produce too much cortisone. Those who have read this book carefully will know which drug is meant: Cytadren. Since it reduces the cortisone level extremely well athletes usually take it directly after completion of a steroid treatment (see also Cytadren). Several athletes take thyroid hormones in this phase since they have an anabolic effect when taken in small dosages and for not excessively long intake intervals. Their effect can be clearly increased by the anticatabolic effect of Clenbuterol which explains why this combination is used during the phase of discontinuance. The use of growth hormone also makes sense since it has a strong anticatabolic/anabolic effect. You can forget Ornithin and Arginin which supposedly increase the realising of GH, because they are ineffective. Distance yourself from the thought that pharmaceutically improved muscle mass can be maintained with "natural methods."
