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ECA yhomibine stack dosages?
- Thread starter tommboy
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todoveritas
New member
What is the anecdotal scoop, Omega? I've been stacking ECA with Yohimbine for a month now and I haven't noticed anything amiss...Am I missing something?
Ephedrine is god, worship it as such.
todoveritas
New member
tommboy said:
My pre-contest stack which I run two weeks at a time alternating with clen is like this:
0600 Aspirin 325 mg (one reg tablet); Ephedrine 25 mg; Caffeine 200 mg; Yohimbine 5 mg
1400 Ephedrine 25 mg; Caffeine 200 mg
Some people don't like the aspirin but I do. It just seems like I sweat more with it than without it. Also, being older, I like the cardiovascular benefits and the anti-arthritic benefits.
Mavafanculo
New member
OMEGA said:
assumming you're healthy, they're actually synergistic - if I recall the yohimbe shuts down your body's thermal regulation system so it cant lower your body temperature as it soars with the ECA oollloolloloo
mentally the yohimbe makes you alittle anxious/paranoid (documented and personal experience), the ephedrine just wires you but no paranoia/anxiety/etc
DJLegacy2k1
New member
I was taking a stack of 25mg E, 100mg C, 100 A, 5mg Y, for 10 weeks and it seemed to work pretty well.
-Legacy
-Legacy
todoveritas
New member
Hi Tommboy. I was using military time: 0600 = 6AM; 1400 = 2 PM. I take these one hour before my morning and afternoon work-outs. It's generally all I need bc I sweat all day with this twice a day dosing. I've lost 23 lbs in the last 6 weeks so it's definitely helping.
todoveritas
New member
In a nutshell:
Catecholamines are neurotransmitters such as epinephrine and norepinephrine that attach to alpha and beta receptors and speed up metabolism. Part of this process involves breakdown of fat in stored areas (adipolysis) which involves stimulation of the B3 receptor in adipose tissue.
Yohimbine is thought to be an alpha-receptor stimulator that increases the sensitivity of the B3 receptor, thus increasing fat-burining efficiency.
Aspirin works a little more mysteriously. It essentially blocks prostaglandins through the inhibition of an enzyme called cyclo-oxygenase. Of the many types of prostaglandins, it is thought to block those that are trying to turn "off" the thermogenic effects of the ephedrine and in so doing, potentiate the fat-burning effect of E.C.A.
Haters will say ASA could also block some prostaglandins involved in muscle-building, (but in a A.A.S.-enriched environment, I say this effect, if present, is negligible).
Catecholamines are neurotransmitters such as epinephrine and norepinephrine that attach to alpha and beta receptors and speed up metabolism. Part of this process involves breakdown of fat in stored areas (adipolysis) which involves stimulation of the B3 receptor in adipose tissue.
Yohimbine is thought to be an alpha-receptor stimulator that increases the sensitivity of the B3 receptor, thus increasing fat-burining efficiency.
Aspirin works a little more mysteriously. It essentially blocks prostaglandins through the inhibition of an enzyme called cyclo-oxygenase. Of the many types of prostaglandins, it is thought to block those that are trying to turn "off" the thermogenic effects of the ephedrine and in so doing, potentiate the fat-burning effect of E.C.A.
Haters will say ASA could also block some prostaglandins involved in muscle-building, (but in a A.A.S.-enriched environment, I say this effect, if present, is negligible).
todo, that was a very informative response and I wish that type of response was the norm here instead of the usual disinformation that is given out.
Out of all these components ephedrine, yohimbe hcl, caffine, aspirin and even albuterol and clenbuterol which ones will have or have the most down regulation in the mechanism in which they work? i.e. how long can you cycle them for and how does it effect weight gain when you stop them. For example when after a cycle of eca does fat come back quicker because whatever fatburning mechanism eca stack was stimulating is now slower and down regulated, how long will that last?
Out of all these components ephedrine, yohimbe hcl, caffine, aspirin and even albuterol and clenbuterol which ones will have or have the most down regulation in the mechanism in which they work? i.e. how long can you cycle them for and how does it effect weight gain when you stop them. For example when after a cycle of eca does fat come back quicker because whatever fatburning mechanism eca stack was stimulating is now slower and down regulated, how long will that last?
todoveritas
New member
tommboy said:
Because you are speaking of adrenergic receptors, the ones which will down regulate fastest are those which most directly stimulate the beta receptors. This would be: clenbuterol > albuterol > ephedrine > caffeine > yohimbine > aspirin. Two weeks is the practical norm for which maximum effectiveness is maintained. That is why I do clen X 2 weeks; alternated with E.C.A. X 2 weeks.
