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(Contradictory Articles) Oaxandrolone Improves or impairs Liver Function?
- Thread starter enzymus
- Start date
S
Stew Meat
Guest
To my knowlege, no research has ever shown it to be hepatoxic. There have been people to claim this, but never has any research been published against it. It is actually used to treat liver damage.
-Stew
-Stew
S
Stew Meat
Guest
Actually, I believe dosses of up to 80mg/day have shown no hepatoxicity.
-Stew
-Stew
Stew is going to rip into me(as usual), but Ulter&myself
had a discussion on this topic a couple of months
back.
Ox is used to treat alcoholics?? Yes.
That doesn't prove anything.
Alcohol induces liver damage if drunk for
extended periods of time.
Now, round some alcoholics, sober them up
and start giving them Ox(a less hepatotoxic
compound than alcohol(well, at the levels they
were drinking at), and OF COURSE LIVER
FUNCTION IS GOING TO IMPROVE.
You've just substituted alcohol for a less hepatotoxic
compoundi.e. Oxandrolone
There's too much contradictory information on the subject.
Is Anavar hepatotoxic? Probably, if used for extended
periods.
It is the least hepatotoxic of all 17-aa's though.
I can't post any studies because I'm using WebTv
(hence the funky margins), so if you guys could post your
studies concerning the hepatotoxicity of Anavar
I'd appreciate as the subject has had me thinking and
a discussion on the subject would be great(scientific)
Where's ulter when you need him? LOL
Godspeed
had a discussion on this topic a couple of months
back.
Ox is used to treat alcoholics?? Yes.
That doesn't prove anything.
Alcohol induces liver damage if drunk for
extended periods of time.
Now, round some alcoholics, sober them up
and start giving them Ox(a less hepatotoxic
compound than alcohol(well, at the levels they
were drinking at), and OF COURSE LIVER
FUNCTION IS GOING TO IMPROVE.
You've just substituted alcohol for a less hepatotoxic
compoundi.e. Oxandrolone
There's too much contradictory information on the subject.
Is Anavar hepatotoxic? Probably, if used for extended
periods.
It is the least hepatotoxic of all 17-aa's though.
I can't post any studies because I'm using WebTv
(hence the funky margins), so if you guys could post your
studies concerning the hepatotoxicity of Anavar
I'd appreciate as the subject has had me thinking and
a discussion on the subject would be great(scientific)
Where's ulter when you need him? LOL
Godspeed
S
Stew Meat
Guest
Will Anavar harm your liver? No
Is Fonz full of shit? Yes
Are anabolic steroids given to alcoholics to sober them up? No
Are there any conflicting studies showing Anavar to be hepatoxic? NO
Is there any reason at all to doubt all the studies that have been done to prove that Anavar will not impair liver function? Yes: Fonz says so.
-Stew
Is Fonz full of shit? Yes
Are anabolic steroids given to alcoholics to sober them up? No
Are there any conflicting studies showing Anavar to be hepatoxic? NO
Is there any reason at all to doubt all the studies that have been done to prove that Anavar will not impair liver function? Yes: Fonz says so.
-Stew
Bonkovsky HL, Fiellin DA, Smith GS, Slaker DP, Simon D, Galambos JT
Department of Medicine, Emory University School of Medicine, Atlanta, Georgia.
The present studies were designed to provide careful measures of effects of oxandrolone, an anabolic steroid, intravenous nutritional supplementation, and the combination of these two treatments on liver functions, metabolic balances, nitrogen metabolism, and nutritional status in patients with moderate to severe alcoholic hepatitis. Of 43 patients originally recruited, 39 (19 men, 20 women) with typical clinical and laboratory features of alcoholic hepatitis (11 Child's-Pugh class B; 28 class C) were admitted to a metabolic unit and completed a 35-day three-phase protocol. Phase I was a 10-day baseline period of observation, during which routine and special quantitative tests of liver function (galactose and antipyrine metabolism), a 7-day elemental balance study, and a 15N, 13C-leucine metabolism study were done. Phase II was a 21-day treatment period during which patients were randomly assigned to receive one of four regimens: 1) standard therapy, consisting of abstinence, a balanced, nutritionally adequate diet, and multivitamins; 2) oxandrolone (20 mg orally four times a day) plus standard therapy; 3) nutritional supplementation, consisting of 2 L daily of 3.5% crystalline amino acids (in 5% dextrose), given by peripheral vein; or 4) a combination of oxandrolone and nutritional supplementation, along with standard therapy. Metabolic balances were repeated during phase II. Phase III was 2 or 3 days posttreatment, during which special studies of liver functions and volumes and leucine metabolism were repeated. All patients who completed phase I of study and were randomly allocated to one of the four treatment groups completed the subsequent two phases. Overall, with time, patients showed highly significant improvements in most clinical and laboratory features. For most standard laboratory tests (e.g., serum albumin, transferrin, prothrombin time) improvements were more marked in patients treated with nutritional supplementation and/or oxandrolone than in those given standard therapy alone. Liver volumes fell in all treatment groups, with greater improvement in those treated with nutritional supplementation. Improvements in galactose and antipyrine metabolism rates were significant only in those treated with nutritional supplementation or oxandrolone. Effects of treatments on metabolic balances, nitrogen metabolism, and measures of nutrition are described in this issue in a companion paper. We conclude that the addition of nutritional supplementation and oxandrolone to standard therapy of moderately severe or severe alcoholic hepatitis is well tolerated, and leads to more rapid improvement in the laboratory parameters measured.
Department of Medicine, Emory University School of Medicine, Atlanta, Georgia.
The present studies were designed to provide careful measures of effects of oxandrolone, an anabolic steroid, intravenous nutritional supplementation, and the combination of these two treatments on liver functions, metabolic balances, nitrogen metabolism, and nutritional status in patients with moderate to severe alcoholic hepatitis. Of 43 patients originally recruited, 39 (19 men, 20 women) with typical clinical and laboratory features of alcoholic hepatitis (11 Child's-Pugh class B; 28 class C) were admitted to a metabolic unit and completed a 35-day three-phase protocol. Phase I was a 10-day baseline period of observation, during which routine and special quantitative tests of liver function (galactose and antipyrine metabolism), a 7-day elemental balance study, and a 15N, 13C-leucine metabolism study were done. Phase II was a 21-day treatment period during which patients were randomly assigned to receive one of four regimens: 1) standard therapy, consisting of abstinence, a balanced, nutritionally adequate diet, and multivitamins; 2) oxandrolone (20 mg orally four times a day) plus standard therapy; 3) nutritional supplementation, consisting of 2 L daily of 3.5% crystalline amino acids (in 5% dextrose), given by peripheral vein; or 4) a combination of oxandrolone and nutritional supplementation, along with standard therapy. Metabolic balances were repeated during phase II. Phase III was 2 or 3 days posttreatment, during which special studies of liver functions and volumes and leucine metabolism were repeated. All patients who completed phase I of study and were randomly allocated to one of the four treatment groups completed the subsequent two phases. Overall, with time, patients showed highly significant improvements in most clinical and laboratory features. For most standard laboratory tests (e.g., serum albumin, transferrin, prothrombin time) improvements were more marked in patients treated with nutritional supplementation and/or oxandrolone than in those given standard therapy alone. Liver volumes fell in all treatment groups, with greater improvement in those treated with nutritional supplementation. Improvements in galactose and antipyrine metabolism rates were significant only in those treated with nutritional supplementation or oxandrolone. Effects of treatments on metabolic balances, nitrogen metabolism, and measures of nutrition are described in this issue in a companion paper. We conclude that the addition of nutritional supplementation and oxandrolone to standard therapy of moderately severe or severe alcoholic hepatitis is well tolerated, and leads to more rapid improvement in the laboratory parameters measured.
Hugh Gellatts
New member
anabolic steroids are given to boozers? Much potential here....
"Hi, my name is Hugh and I'm an alcoholic. Now give me my fucking bottle of Anavar!"
"Hi, my name is Hugh and I'm an alcoholic. Now give me my fucking bottle of Anavar!"
