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testoman79
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testoman79 said:
Unfortunately, the anabolic effects of Clenbuterol were never proven in humans. The study which that conclusion was based upon was performed with rodents. The good news is that Albuterol has in fact been linked to hypertrophy in humans.
Oral albuterol dosing during the latter stages of a resistance exercise program.
J Strength Cond Res. 2005 Feb;19(1):102-7.
Caruso JF, Hamill JL, De Garmo N.
Healthcare Research Associates Inc., Orlando, Florida 32819.
Caruso, J.F., J.L. Hamill, and N. De Garmo. Oral albuterol dosing during the latter stages of a resistance exercise program. J. Strength Cond. Res. 19(1):102-107. 2005.-Subjects performed isoload variable resistance exercise (REX) 3 days per week. After 10 weeks, they received a double-blind albuterol (n = 11) or placebo (n = 11) capsule assignment with no crossover and continued training. During the first week of capsule administration, dosages were increased from 4 mg to 16 mg daily and then maintained for 14 days. At weeks 0, 10, and 13, we measured upper arm and thigh cross-sectional area, knee and elbow extensor and flexor (KE, KF, EE, EF) strength at 3 angular velocities, and lean body mass. Data after 10 weeks showed insignificant between-group differences. From weeks 10-13, as subjects continued REX training, albuterol evoked higher (p < 0.05) KE-KF strength gains at multiple velocities versus placebo dosing. A higher lean body mass trend also occurred with albuterol from weeks 10-13. Results suggest that albuterol augments REX to provide greater strength gains from hypertrophic factors than an REX-placebo assignment.
Jenetic
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testoman79
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Excellent reply Jenetic you are the man!!!!
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testoman79
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If your going to use it with your pct i would suggest 4 - 5 tablets ED until finish pct i was thinking of trying this but have not as yet. Jenetic will probably be able to advise you better than I, take a look at the statement below this will explain how some think Clen can help in pct.
All this, however, helps only if the athlete is able to mostly block out the catabolic effect of the increased cortisone level. A compound which, because of its distinct anticatabolic effect, fulfills this requirement is the beta-2 sympathomimetic, Clenbuterol. Clenbuterol successfully blocks the cortisone receptors so that the athlete is usually able to maintain a large portion of the strength and muscle mass built up by the steroids. The intake of Clenbuterol begins directly at the end of the steroid therapy and continues over 8-10 weeks (see also Clenbuterol). Another compound of the group of sympaticomimetics which also has an anticatabolic effect (but less pronounced than Clenbuterol) is Ephedrine. Probably the most suitable drug in this situation is a preparation which in school medicine is used in the treatment of the Cushing's syndrome, a hyperfunction of the adrenal glands which causes the body to produce too much cortisone. Those who have read this book carefully will know which drug is meant: Cytadren. Since it reduces the cortisone level extremely well athletes usually take it directly after completion of a steroid treatment (see also Cytadren). Several athletes take thyroid hormones in this phase since they have an anabolic effect when taken in small dosages and for not excessively long intake intervals. Their effect can be clearly increased by the anticatabolic effect of Clenbuterol which explains why this combination is used during the phase of discontinuance. The use of growth hormone also makes sense since it has a strong anticatabolic/anabolic effect. You can forget Ornithin and Arginin which supposedly increase the realising of GH, because they are ineffective. Distance yourself from the thought that pharmaceutically improved muscle mass can be maintained with "natural methods
Hope this will help!.
All this, however, helps only if the athlete is able to mostly block out the catabolic effect of the increased cortisone level. A compound which, because of its distinct anticatabolic effect, fulfills this requirement is the beta-2 sympathomimetic, Clenbuterol. Clenbuterol successfully blocks the cortisone receptors so that the athlete is usually able to maintain a large portion of the strength and muscle mass built up by the steroids. The intake of Clenbuterol begins directly at the end of the steroid therapy and continues over 8-10 weeks (see also Clenbuterol). Another compound of the group of sympaticomimetics which also has an anticatabolic effect (but less pronounced than Clenbuterol) is Ephedrine. Probably the most suitable drug in this situation is a preparation which in school medicine is used in the treatment of the Cushing's syndrome, a hyperfunction of the adrenal glands which causes the body to produce too much cortisone. Those who have read this book carefully will know which drug is meant: Cytadren. Since it reduces the cortisone level extremely well athletes usually take it directly after completion of a steroid treatment (see also Cytadren). Several athletes take thyroid hormones in this phase since they have an anabolic effect when taken in small dosages and for not excessively long intake intervals. Their effect can be clearly increased by the anticatabolic effect of Clenbuterol which explains why this combination is used during the phase of discontinuance. The use of growth hormone also makes sense since it has a strong anticatabolic/anabolic effect. You can forget Ornithin and Arginin which supposedly increase the realising of GH, because they are ineffective. Distance yourself from the thought that pharmaceutically improved muscle mass can be maintained with "natural methods
Hope this will help!.
mrplunkey said:
Effective dosages start as low as 4 mgs ED and venture as high as 20 mgs ED with no adverse effects. Personally, I prefer the higher dosage range of 16-20 mgs ED, but many people find 4-8 mgs to be sufficient. Start with a lower dosage and asses your tolerance. In regards to stacking with Ketotifen, it is beneficial in the same manner as stacking it with Clenbuterol. 1 mg Ketotifen ED taken at bedtime is plenty.
Jenetic
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