Please Scroll Down to See Forums Below
napsgear
genezapharmateuticals
domestic-supply
puritysourcelabs
UGL OZ
UGFREAK
napsgeargenezapharmateuticals domestic-supplypuritysourcelabsUGL OZUGFREAK

Being Shut Down

bbforlife

New member
Ok so when we are talkin aobut "being shut down" after cycle. We are talking about are nuts shrinking, not getting it up and low test levels ,. . correct. So on cycles that involve tren ,you have the potiental to be shut down pretty hard? What causes this? Scientifically, lol
thanks bros
 
To me shut-down is low test levels. Symptoms are: loss of size, hardness, low energy and low sexdrive. I have never not been able to get it up or had any substantial nutshrinkage. Fina shuts some down hard, others not. Same with deca. Reasons for fina: progesterone effect?, strong receptor binder? Reasons for deca: progesterone effect?, extremely long acting effect? There are no definitive answers on why these two supposedly shut you down harder.
 
Your body monitors many hormone levels and adjusts its natural output accordingly. Estrogen is one of the primary feedback mechanisms for test production and when you use something like an anti-e and reduce extrogen your body will up test production in an effort to raise estrogen. Howevery there are usually more than one feedback mechanism and in addition to monitoring estrogen the body also monitors androgen levels so stopping E production will only increase test to a certain degree until the body ceases to increase test production any more. On cycle androgen levels are so high it causes almost complete shutdown of natural test production, some AAS more than others and their are many factors which come into play.
 
Zyg, nice job explaining that.

If you do HCG and clomid your natural levels will come back faster.
Being ok is a broad term. There will always be a lapse from being on/ coming off and getting back to normal. Each individual varies with this time frame.
 
bbforlife said:
so as long as you run your hcg ,clomid at the end you should be ok?

HCG is a glycoprotien that mimics our bodies own LH, having said that and we know that the body will reduce output of a hormone that is introduces externally taking HCG will work at returning the size and reversing atrophy to the testis since it mimics LH. However there is a down side and that since we are getting LH externally via HCG our body will not need to produce its own. With that in mind, I personally feel the best time to use HCG is at the tail end of a cycle of short esters, or just post cycle with long esters. Taking HCG while androgen levels are too high for the body to recover will reverse atrophy so that once androgen levesl diminish post cycle your testis are in a better position to start producing on their own. However LH release is what gets the testis going and since you have been on cycle LH level have also been suppressed so regardless of what you take you still have to wait for the body to respond and start producing LH again.

Clomid/nolva etc blocks the ER and the idea is that making the body think E is lower than it is will force an overcompensation of LH release to speed recovery. For some it works well and for others it doesnt seem to matter.

For me I notice that running ana/let or aromasin past end of cycle speeds recovery.
 
Top Bottom