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Those aas that are said to be strong androgens often have low binding affinity to the androgen receptor.
And the opposite is also true, primo, deca, anavar have strong binding affinity but are said to be less androgenic.
Could we conclude that many of the androgen effects are from non-AR pathways?
If so, are all of the androgen effects from both AR and non-AR effects?
Does all here agree there are non-AR anabolic effects as well?
Any thoughts are welcome.
Qrios
And the opposite is also true, primo, deca, anavar have strong binding affinity but are said to be less androgenic.
Could we conclude that many of the androgen effects are from non-AR pathways?
If so, are all of the androgen effects from both AR and non-AR effects?
Does all here agree there are non-AR anabolic effects as well?
Any thoughts are welcome.
Qrios
