But Clomid is not medically proven for anything other than as a female fertility drug.
C'mon Nelson, you know this isn't true. The entire goal of PCT is to get to the pituitary to release LH into the bloodstream so that the testicles produce testosterone again and the HPTA can reach an equilibrium.
We use the term "shut down" as a euphemism meaning the brain has stopped or lowered the production of LH (due to the presence of steroids) which means our testicles aren't receiving the proper signaling to produce sufficient natural T.
Below are just a couple of medical studies that illustrate the effectiveness of clomid at stimulating the release of LH. The first study even documents this result in 100% of men (out of 178 patients).
Int J Impot Res. 2003 Jun;15(3):156-65.
Clomiphene increases free testosterone levels in men with both secondary hypogonadism and erectile dysfunction: who does and does not benefit?
Guay AT, Jacobson J, Perez JB, Hodge MB, Velasquez E.
Center for Sexual Function (Endocrinology), Peabody, Massachusetts 01960, USA.
[email protected]
Abstract
Secondary hypogonadism is more common than primary gonadal failure and is seen in chronic and acute illnesses. Although testosterone has a role in erections, its importance in erectile dysfunction (ED) has been controversial. Hypogonadism produced by functional suppression of pituitary gonadotropins has been shown to correct with Clomiphene citrate, but with a modest effect on sexual function. We wondered if longer treatment would produce improved results. A total of 178 men with secondary hypogonadism and ED received Clomiphene citrate for 4 months. Sexual function improved in 75%, with no change in 25%, while
significant increases in luteinizing hormone (P<0.001) and free testosterone (P<0.001) occurred in all patients. Multivariable analysis showed that responses decreased significantly with aging (P<0.05). Decreased responses also occurred in men with diabetes, hypertension, coronary artery disease, and multiple medication use. Since these conditions are more prevalent with aging, chronic disease may be a more important determinant of sexual dysfunction. Men with anxiety-related disorders responded better to normalization of testosterone. Assessment of androgen status should be accomplished in all men with ED. For those with lower than normal age-matched levels of testosterone treatment directed at normalizing testosterone with Clomiphene citrate is a viable alternative to giving androgen supplements.
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J Clin Endocrinol Metab. 1995 Dec;80(12):3546-52.
Effect of raising endogenous testosterone levels in impotent men with secondary hypogonadism: double blind placebo-controlled trial with Clomiphene citrate.
Guay AT, Bansal S, Heatley GJ.
Section of Endocrinology, Lahey Clinic, Burlington, Massachusetts 01805, USA.
Abstract
Secondary hypogonadism is not an infrequent abnormality in older patients presenting with the primary complaint of erectile dysfunction. Because of the role of testosterone in mediating sexual desire and erectile function in men, these patients are usually treated with exogenous testosterone, which, while elevating the circulating androgens, suppresses gonadotropins from the hypothalamic-pituitary axis. The response of this form of therapy, although extolled in the lay literature, has usually not been effective in restoring or even improving sexual function. This failure of response could be the result of suppression of gonadotropins or the lack of a cause and effect relationship between sexual function and circulating androgens in this group of patients. Further, because exogenous testosterone can potentially increase the risk of prostate disease, it is important to be sure of the benefit sought, i.e. an increase in sexual function. In an attempt to answer this question, we measured the hormone levels and studied the sexual function in 17 patients with erectile dysfunction who were found to have secondary hypogonadism. This double blind, placebo-controlled, cross-over study consisted of treatment with Clomiphene citrate and a placebo for 2 months each.
Similar to our previous observations, LH, FSH, and total and free testosterone levels showed a significant elevation in response to Clomiphene citrate over the response to placebo. However, sexual function, as monitored by questionnaires and nocturnal penile tumescence and rigidity testing, did not improve except for some limited parameters in younger and healthier men. The results confirmed that there can be a functional secondary hypogonadism in men on an out-patient basis, but correlation of the hormonal status does not universally reverse the associated erectile dysfunction to normal, thus requiring closer scrutiny of claims of cause and effect relationships between hypogonadism and erectile dysfunction.
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