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How toxic is winny to the liver

General-K

New member
OK bro`s I know winny is toxic to your liver and whilst i run it i am going to take milk thistle ED to help detox my liver but can an 8 week cycle of winny & test prop winny at 50mg daily and test prop at 600mg per week cause me to have serious liver problems any comments appreciated i have researched this but nothing seems to tell me exactly how bad (toxic) it is I have never had any liver problems before.
 
I was running winny orals for 3 weeks at 50mg/day and went to the doc for a blood test, and he called me and said that we needed to be a little worried about my elevated liver enzymes. Of course, I wasn't taking anything to help my situation at that time, but it was the cause for a little concern. I would run it the first 6 weeks if I were you.
 
i think it was bruce who said he had a friend who was on winni year round and had perfectly normal liver tests.....guess it effects each person differently....def take milk thistle or tylers liver detox though
 
ricorico said:
i think it was bruce who said he had a friend who was on winni year round and had perfectly normal liver tests.....guess it effects each person differently....def take milk thistle or tylers liver detox though

I would luv to know what his before and after pic looked
 
milk thistle isn't going to do shit... keep your money instead. btw, it's not very toxic, you might have slightly elevated liver enzymes, but it's no big deal.
 
Everyone is different. Winny is a 17aa and so can be liver toxic dependant on may factors that are common to your body.

The only way to tell is to have your liver enzyme count checked regularily.

I would treat with caution in terms of dosage and length of time used not only for your liver but also your joints

Wrongun!
 
General-K said:
OK bro`s I know winny is toxic to your liver and whilst i run it i am going to take milk thistle ED to help detox my liver but can an 8 week cycle of winny & test prop winny at 50mg daily and test prop at 600mg per week cause me to have serious liver problems any comments appreciated i have researched this but nothing seems to tell me exactly how bad (toxic) it is I have never had any liver problems before.

All orals (except andriol) are 17aa and, correct me if I'm wrong but, none of them are anymore "liver-toxic" than the others. The level of toxicity hinges on the number of mg's used per day. Thus, 50mg of anadrol is no more liver-toxic than 50mg of winstrol, it's just that when people are using anadrol they tend to take more mg per day than they do when they're taking dbol.

I find it very interesting that some people will advocate 100mg (and even 150mg) per day of anadrol but if someone asked a question about taking 150mg of dbol per day, those same people would tell them "you're nuts, you're going to rot out your liver". I ask you, what's the diff??
 
Of course some are more liver toxic than others. It has as much to do with the chemical itself as it does the fact that's it been made 17aa.
Anadrol is not much different than dbol. But Oxandrolone is significantly different than the other two.
And just to be clear, the liver toxicity levels of AS are CLEARLY overstated. Mainly because of poor testing methods on weight training subjects by the medical community over the last several years.

Clin J Sport Med. 1999 Jan;9(1):34-9. Related Articles, Links


Anabolic steroid-induced hepatotoxicity: is it overstated?

Dickerman RD, Pertusi RM, Zachariah NY, Dufour DR, McConathy WJ.

The Department of Biomedical Science, University of North Texas Health Science Center, Fort Worth 76107-2699, USA.

OBJECTIVE: There have been numerous reports of hepatic dysfunction secondary to anabolic steroid use based on elevated levels of serum aminotransferases. This study was conducted to distinguish between serum aminotransaminase elevations secondary to intense resistance training and anabolic steroid-induced hepatotoxicity in elite bodybuilders. DESIGN: This was a case-control study of serum chemistry profiles from bodybuilders using and not using anabolic steroids with comparisons to a cohort of medical students and patients with hepatitis. PARTICIPANTS: The participants were bodybuilders taking self-directed regimens of anabolic steroids (n = 15) and bodybuilders not taking steroids (n = 10). Blood chemistry profiles from patients with viral hepatitis (n = 49) and exercising and nonexercising medical students (592) were used as controls. MAIN OUTCOME MEASURES: The focus in blood chemistry profiles was aspartate aminotransferase (AST), alanine aminotransferase (ALT), gamma-glutamyltranspeptidase (GGT), and creatine kinase (CK) levels. RESULTS: In both groups of bodybuilders, CK, AST, and ALT were elevated, whereas GGT remained in the normal range. In contrast, patients with hepatitis had elevations of all three enzymes: ALT, AST, and GGT. Creatine kinase (CK) was elevated in all exercising groups. Patients with hepatitis were the only group in which a correlation was found between aminotransferases and GGT. CONCLUSION: Prior reports of anabolic steroid-induced hepatotoxicity based on elevated aminotransferase levels may have been overstated, because no exercising subjects, including steroid users, demonstrated hepatic dysfunction based on GGT levels. Such reports may have misled the medical community to emphasize steroid-induced hepatotoxicity when interpreting elevated aminotransferase levels and disregard muscle damage. For these reasons, when evaluating hepatic function in cases of anabolic steroid therapy or abuse, CK
 
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